Magnetic toroidal core having successive windings with the pair of lead ends of eachwinding spaced from the pair of lead ends of each other winding

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  机译： 带状材料连续卷绕在铁芯上-在公共轴上有一对隔开的端柱和滚子支撑臂

• 4. Method for establishing winding lead wires for windings supported on magnetic cores [P] . 美国专利： US3879841A . 1975-04-29

  机译： 用于磁芯上支撑的绕组的绕组导线的建立方法

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  机译： 对铅丝的布置及其连接方法，以及使用该引线丝制造旋转机的绕线方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. A Note on Pad Approximants Pairs as Limits of Algebraic Polynomials Pairs of Weighted Best Approximation in Orlicz Spaces [J] . F.E.Levis . 分析理论与应用：英文刊 . 2015,第3期
• 3. Space Magnetism and Superconductivity: Diamagnetic Expulsion, Meissner Effect, Magnetic Pressure and Quantum Trapping Lead to the Origin and Stability of the Saturn Rings [J] . Vladimir V. Tchernyi (Cherny) ,Andrey Yu. Pospelov ,Sergey V. Kapranov . 应用数学与应用物理（英文） . 2019,第8期
• 4. 特高压变压器绕组介损不拆高压引线试验方法研究 [C] . 张明兴 ,张国栋 ,黄斯茜 . 第十三届全国变压器技术学术年会 . 2017
• 5. 高压直流超导限流器绕组和电流引线绝缘研究 [A] . 邢云琪 . 2017