method and device for determining the concentration of a line stroemenden spoils a suspension of dredging sludge with water

• 1. 确定纸浆悬浮液中纤维浓度或粘度的转矩或剪切力传递装置和复位转矩或剪切力传递器中的传递轴的方法 [P] . 中国专利： CN100516798C . 2009.07.22
• 2. 确定纸浆悬浮液中纤维浓度或粘度的转矩或剪切力传递装置和复位转矩或剪切力传递器中的传递轴的方法 [P] . 中国专利： CN1890544A . 2007-01-03
• 3. method and device for determining the concentration of a line stroemenden spoils a suspension of dredging sludge with water [P] . 德国专利： DE000001773050A . 1971-09-23

  机译： 确定管线浓度的方法和装置用水破坏挖泥污泥的悬浮液

• 4. METHOD AND DEVICE FOR DETERMINING THE CONVEY CONCENTRATION OF DREDGING SPOIL OF A SUSPENSION OF DREDGING SPOIL AND WATER [P] . 美国专利： US3554011A . 1971-01-12

  机译： 测定悬浮泥沙和水悬浮泥沙浓度的方法和装置

• 5. METHOD AND APPARATUS FOR DETERMINING THE CONCENTRATION OF DREDGER SPOIL IN A SUSPENSION OF DREDGER SPOIL AND WATER FLOWING THROUGH A PIPE [P] . 英国专利： GB1217579A . 1970-12-31

  机译： 测定悬浮物中悬浮物的浓度和流经管道的水的方法和装置

• 1. Development and application of diffusive gradients in thin films for determining inorganic arsenic concentrations in natural waters [J] . 中国地球化学学报（英文版） . 2006,第0z1期
• 2. Development and application of diffusive gradients in thin films for determining inorganic arsenic concentrations in natural waters [J] . Jared, G., Panther, 中国地球化学学报：英文版 . 2006,第B08期
• 3. Comparison of Geotextiles by Aeration Vacuum Rapid Dewatering Technique on Dredged Sludge [J] . Yuan Zhou, Jie Bian, Hui Tao, 哈尔滨工业大学学报（英文版） . 2016,第004期
• 4. Use of QSAR and SSD methods on deriving predicted no-effect concentrations in seawater and sediment for ten individual parent- and alkyl-PAHs and a case study on the assessment of their ecological risks from the Dalian Bay, China [J] . Ying Wang, Xing Liu, Yi Cong, 海洋学报（英文版） . 2020,第012期
• 5. Optical method of determining the concentration of suspension in natural waters [C] . Barbara Pawlak, Jadwiga Mrozek-Lejman, Radoslaw Gasowski Slovak-Czech-Polish Conference on Wave and Quantum Aspects of Contemporary Optics . 2005
• 6. Impact of secondary treatment types and sludge handling processes on estrogen concentration in wastewater sludge. [D] . Marti, Erica. 2012
• 7. Intercomparison study on commonly used methods to determine microplastics in wastewater and sludge samples [O] . Mirka Lares, Mohamed Chaker Ncibi, Markus Sillanpää, -1
• 8. The marine disposal of sewage sludge and dredge spoil in the waters of the New York Bight [O] . Ralph A. Horne, A. J. Mahler, R. C. Rossello 1971

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 提高射流式挖泥装置排泥浓度的试验研究 [C] . 龙新平 ,朱劲木 ,陆宏圻 . 2002全国流体机械技术会议 . 2002
• 3. 饮用水中溴酸盐浓度检测方法研究 [A] . 庾志衡 . 2011