METHOD OF AND DEVICE FOR PRODUCING AND PROTECTING DEPOSITS OF SEDIMENTARY MATERIAL ON FLOORS OF BODIES OF WATER

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• 2. 沉积物去除装置和方法，沉积物去除系统 [P] . 中国专利： CN108057693A . 2018-05-22
• 3. A METHOD OF AND DEVICE FOR PRODUCING AND PROTECTING AND PROTECTING DEPOSITS OF SEDIMENTARY MATERIAL ON THE FLOOR OF BODIES OF WATER. [P] . IN134171B . 1974-11-30

  机译： 在水体上生产，保护和沉积沉积物的方法和装置。

• 4. A METHOD OF AND DEVICE FOR PRODUCING AND PROTECTING DEPOSITS OF SEDIMENTARY MATERIAL ON THE FLOOR OF BODIES OF WATER [P] . IL38522D0 . 1972-03-28

  机译： 在水体底面上生产和保护沉积物的方法和装置

• 5. A METHOD OF AND DEVICE FOR PRODUCING AND PROTECTING DEPOSITS OF SEDIMENTARY MATERIAL ON THE FLOOR OF BODIES OF WATER [P] . ZA72122B . 1972-11-29

  机译： 在水体底面上生产和保护沉积物的方法和装置

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• 2. The Hajjar deposit of Guemassa (SW Meseta, Morocco): a metamorphosed syn-sedimentary massive sulphide ore body of the Iberian type of volcano-sedimentary massive sulphide deposits [C] . C. Marignac, C. Marignac, M. Hibti Biennial SGA-SEG meeting . 2001
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• 5. DESIGN AND MANUFACTURE OF AN ACCELERATING DEVICE FOR STAINS ON POLYMERIC FLOOR MATERIALS : Development of a method of evaluation for the state of stains on polymeric floor finishing materials (Part 2) [O] . HIDENORI ONO, HIROSHI BABA, MAKOTO YOSHIOKA 1984
• 6. Development of Novel Processing Methods for Producing Bone Augmentation and Replacement Materials and Devices [R] . Campbell, A. A. , Armstrong, B. 1999

• 1. QUANTUM-CHEMICAL MODIFICATIONS OF SURFACE:NEW METHODS FOR PROTECTING MATERIALS FROM CORROSION [J] . R. T. Malkhasyan (Scientific Production Enterprise Atom Ministry of Industry and Trade 3/1 Tevosyan Yerevan 375076 Armenia) . 金属学报：英文版 . 2001,第6期
• 2. Applications of Secondary Electron Composition Contrast Imaging Method in Microstructure Studies on Heterojunction Semiconductor Devices and Multilayer Materials [J] . 刘安生 ,邵贝羚 ,安生 . 稀有金属：英文版 . 1999,第2期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. Preparation of Wastewater Phosphorus Removal Materials by Dry FGD Ash Produced from Iron Industry [J] . LIN Liang . 结构化学 . 2013,第002期
• 5. 硝基苯酚在黄河水体沉积物上的吸附行为研究 [C] . 李改枝 ,哈斯其木格 ,刘颖 . 华北地区五省市化学学术研讨会 . 2001
• 6. 沉积物钙形态分析及菹草—沉积物—上覆水体系中钙—磷关系研究 [A] . 何君 . 2013