Method and apparatus for the determination of the electron density in a partial volume of a body

• 1. 使用粒子束照射身体或物体中预定最小目标体积的系统、为系统确定计划目标体积的方法和仪器 [P] . 中国专利： CN101663068B . 2012.11.28
• 2. 用于确定吸收性物品中的身体流出物的体积的系统 [P] . 中国专利： CN210834672U . 2020-06-23
• 3. METHOD AND APPARATUS FOR THE DETERMINATION OF ELECTRON DENSITY IN A PART VOLUME OF A BODY [P] . CA1020292A . 1977-11-01

  机译： 测定身体部分体积中电子密度的方法和装置

• 4. METHOD AND APPARATUS FOR THE DETERMINATION OF ELECTRON DENSITY IN A PART VOLUME OF A BODY [P] . 英国专利： GB1466617A . 1977-03-09

  机译： 测定身体部分体积中电子密度的方法和装置

• 5. Method and apparatus for the determination of electron density in a part volume of a body [P] . 美国专利： US3961186A . 1976-06-01

  机译： 用于确定人体部分体积中的电子密度的方法和设备

• 1. Estimating canopy closure density and above-ground tree biomass using partial least square methods in Chinese boreal forests [J] . LEI Cheng-liang, JU Cun-yong, CAI Ti-jiu, 林业研究（英文版） . 2012,第002期
• 2. Determination of Partial Molar Volumes of EPA and DHA Ethyl Esters in Supercritical Carbon Dioxide [J] . 中国化学快报（英文版） . 2002,第001期
• 3. 'Thermodynamic' method for electron density determination by dielectronic satellites to high members of He-like Rydberg seria lines [C] . International colloquium on X-ray lasers . 1990
• 4. Apparatus and method for combined acoustic resonance spectroscopy density determinations. [D] . Tatro, Daniel Scott. 1996
• 5. Mitochondrial and cytoplasmic ribosomes from mammalian tissues. Further characterization of ribosomal subunits and validity of buoyant-density methods for determination of the chemical composition and partial specific volume of ribonucleoprotein particles [O] . Ada Sacchi, Ugo Ferrini, Paola Londei, 1977
• 6. Mitochondrial and cytoplasmic ribosomes from mammalian tissues. Further characterization of ribosomal subunits and validity of buoyant-density methods for determination of the chemical composition and partial specific volume of ribonucleoprotein particles [O] . Sacchi, Ada, Ferrini, Ugo, Londei, Paola, 1977
• 7. Reference Method 24-Determination of Volatile Matter Content, Water Content, Density, Volume Solids, and Weight Solids of Surface Coatings. Reference Method 25-Determination of Total Gaseous Nonmethane Organic Emissions as Carbon. Background Information for Promulgated Test Methods [R] . 1980

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Effect of Flaky Plastic Particle Size and Volume Used as Partial Replacement of Gravel on Compressive Strength and Density of Concrete Mix [J] . Stanley O. Osubor ,Kamoru A. Salam ,Taiwo M. Audu . 环境保护（英文） . 2019,第6期
• 3. Partial and Full Arc Volumetric Modulated Arc Therapy in Lung Cancer Stereotactic Body Radiotherapy with Different Definitions of Internal Target Volume Based on 4D CT [J] . Wu Wang ,Didi Chen ,Ce Han . 医学物理学、临床工程、放射肿瘤学（英文） . 2018,第4期
• 4. 应用于有限体积法中的剪切滑移网格方法 [C] . Feng Xie ,谢峰 ,Kun Qu . 第四届全国非定常空气动力学学术会议 . -1
• 5. 低压成套开关设备可靠性评估中先验分布确定方法的研究 [A] . 王蒙 . 2014