A method in the production of so2 - water, as well as low percent strength so2 - gas from combustion gas with a high so2 - content

• 1. 从SO2含量暂时非常高的气体中除去SO2的方法 [P] . 中国专利： CN110366443A . 2019-10-22
• 2. 用于生产至少一种具有低CO₂含量的气体和至少一种具有高CO₂含量的流体的方法 [P] . 中国专利： CN102497917B . 2014.10.01
• 3. A method in the production of so2 - water, as well as low percent strength so2 - gas from combustion gas with a high so2 - content [P] . 德国专利： DE000001667370C3 . 1976-08-19

  机译： 从高SO2含量的燃烧气体生产SO2-水以及低百分比强度SO2-气体的方法

• 4. A method in the production of so2 - water, as well as low percent strength so2 - gas from combustion gas with a high so2 - content [P] . 德国专利： DE1667370A1 . 1971-06-24

  机译： 从高SO2含量的燃烧气体生产SO2-水以及低百分比强度SO2-气体的方法

• 5. process for simultaneous production of so2 - water and gas with low so2 content of the high so2 content [P] . AT271501B . 1969-06-10

  机译： 同时生产SO2-高SO2含量的低SO2含量的水和气的方法

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• 8. Conteúdo lipídico e composição de ácidos graxos de microalgas expostas aos gases CO2, SO2 e NO Lipid content and fatty acids composition variation of microalgae exposed to CO2, SO2 and NO [O] . Elisangela Martha Radmann, Jorge Alberto Vieira Costa 2008
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 一种测定矿物中包裹体水与气体组分绝对含量的气相色谱方法 [C] . 杨丹 ,崔艳合 ,徐文艺 . 第十四届全国包裹体及地质流体学术研讨会 . 2004
• 3. 燃气成分及其含量对气体机燃烧过程的影响 [A] . 于华 . 2016