Method and device for to separate of the iron, zinc and lead to dust throat or mud throat

• 1. 一种从电炉粉尘中一步回收分离锌铁的方法 [P] . 中国专利： CN113881852A . 2022-01-04
• 2. 一种电炉粉尘制备金属铁及锌/氧化锌的方法 [P] . 中国专利： CN101914685B . 2012.01.25
• 3. Method and device for to separate of the iron, zinc and lead to dust throat or mud throat [P] . 法国专利： FR2336484B1 . 1978-06-23

  机译： 分离铁，锌并导致粉尘或泥喉的方法和装置

• 4. Recovery of zinc, lead and iron - from furnace throat gas dusts, using a hydrocylone or similar separator [P] . 法国专利： FR2299089B1 . 1978-11-10

  机译： 使用水力旋流器或类似的分离器从炉喉气尘中回收锌，铅和铁

• 5. Recovery of zinc, lead and iron - from furnace throat gas dusts, using a hydrocylone or similar separator [P] . 法国专利： FR2299089A1 . 1976-08-27

  机译： 使用水力旋流器或类似的分离器从炉喉气尘中回收锌，铅和铁

• 1. Wetting agent investigation for controlling dust of lead-zinc ores [J] . WU Chao, PENG Xiao-lan, WU Guo-min 中国有色金属学报（英文版） . 2007,第001期
• 2. Volatilization of zinc and lead in direct recycling of stainless steel making dust [J] . PENG Ji(彭及), PENG Bing(彭兵), YU Di(余笛), 中国有色金属学报（英文版） . 2004,第002期
• 3. Removal of cadmium, zinc, copper and lead by red mud, an iron oxides containing hydrometallurgical waste [C] . M. Vaclavikova, P. Misaelides, G. Gallios, International Workshop on New Sources, Novel Phases, New Applications . 2005
• 4. Mouth-throat deposition with non-ballistic pharmaceutical aerosol inhalation devices. [D] . DeHaan, Wesley Hugh. 2002
• 5. Rhinitis Ocular Throat and Dermal Symptoms Headache and Tiredness among Students in Schools from Johor Bahru Malaysia: Associations with Fungal DNA and Mycotoxins in Classroom Dust [O] . Dan Norbäck, Jamal Hisham Hashim, Gui-Hong Cai, -1
• 6. Discussion: “Theoretical Basis for Extrapolation of Calibration Data of PTC 6 Throat Tap Nozzles” (Murdock, J. W., and Keyser, D. R., 1991, ASME J. Eng. Gas Turbines Power, 113, pp. 228–232) and “A Method for the Extrapolation of Calibration Data of PTC 6 Throat Tap Nozzles” (Murdock, J. W., and Keyser, D. R., 1991, ASME J. Eng. Gas Turbines Power, 113, pp. 233–239) [O] . J. Westcott 1991

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. A novel hydrothermal method for zinc extraction and separation from zinc ferrite and electric arc furnace dust [J] . Hui-gang Wang ,Yang Li ,Jian-ming Gao . 矿物冶金与材料学报 . 2016,第002期
• 3. Study on the Evaporation Kinetics of Zinc and Lead in Zn-Pb-Bearing Dust Pellets Mixed with Coal Powder [J] . Wang Dongyan ,Chen Weiqing ,Zhou Rongzhang . 钢铁研究学报：英文版 . 1999,第1期
• 4. 高炉污泥处理用的水力旋流设备——从高炉粉尘中分离富锌分的方法 [C] . Stein Callenfels JE van . 2003中国钢铁年会 . 2003
• 5. 电炉粉尘的锌铁分离及制备Ni--Zn铁氧体的研究 [A] . 陈昌 . 2021