lever to change of torque, because ta from the increase in strength resistant pumps and vehicles esinili souivalenti brake mechanisms

• 1. 利用全权主动燃料管理改变车辆发动机制动转矩的方法 [P] . 中国专利： CN107939536B . 2021.05.14
• 2. 利用全权主动燃料管理改变车辆发动机制动转矩的方法 [P] . 中国专利： CN107939536A . 2018-04-20
• 3. lever to change of torque, because ta from the increase in strength resistant pumps and vehicles esinili souivalenti brake mechanisms [P] . 日本专利： JPS5633253B2 . 1981-08-03

  机译： 杆来改变扭矩，因为ta来自于强度更高的泵和车辆的制动机制

• 4. lever to change of torque, because ta from the increase in strength resistant pumps and vehicles esinili souivalenti brake mechanisms [P] . 法国专利： FR2268300B1 . 1977-10-28

  机译： 杆来改变扭矩，因为ta来自于强度更高的泵和车辆的制动机制

• 5. lever to change of torque, because ta from the increase in strength resistant pumps and vehicles esinili souivalenti brake mechanisms [P] . 日本专利： JPS50146024A . 1975-11-22

  机译： 杆来改变扭矩，因为ta来自于强度更高的泵和车辆的制动机制

• 1. A new braking force distribution strategy for electric vehicle based on regenerative braking strength continuity [J] . LIAN Yu-feng, TIAN Yan-tao, HU Lei-lei, 中南大学学报（英文版） . 2013,第012期
• 2. Strength Analysis and Structural Optimization of Lever of Air Disc Brake [C] . Guoling Wang, Xuexun Guo, Quan Zhou SAE Annual Brake Colloquium and Exhibition . 2014
• 3. Pove?anje ukupnog ko?nog momenta magnetoreolo?ke disk ko?nice primenom kombinovanog re?ima rada = Magnetorheological disk brake overall braking torque increase using combined operating mode [D] . Pozni?, Aleksandar. 2017
• 4. Development and Characterization of a Compact Device for Measuring the Braking Torque of a Vehicle [O] . Ester Olmeda, María Garrosa, Susana Sanz Sánchez, 2020
• 5. Study on Differential Regenerative Braking Torque Control to Increase the Stability of the Small Electric Vehicle With Four In-Wheel Motors [O] . Mohamad Heerwan Peeie, N. M. Ali, Ogino H. 2017
• 6. Torque-tension relationships and static preload loss of high strength, heat resistant, and corrosion resistant aircraft alloy fasteners [R] . Hart, V. G., Jones, J. F. 1971

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Mechanisms Efflux Pumps of iAcinetobacter baumannii/i(MDR): Increasing Resistance to Antibiotics [J] . Francis T. Dongmo Temgoua ,Liang Wu . 生物科学与医学（英文） . 2019,第1期
• 3. A new braking force distribution strategy for electric vehicle based on regenerative braking strength continuity [J] . 廉宇峰 ,田彦涛 ,胡蕾蕾 . 中南大学学报 . 2013,第012期
• 4. Investigation on the strengthening mechanism of S30432 austenitic heat-resistant steel [J] . ZHU Lihui~(1))ZHANG Yujie~(1))WANG Qijiang~(2)) and XU Songqian~(2)) 1) School of Materials Science and EngineeringShanghai UniversityShanghai 200072China 2) Baoshan Iron Steel Co.Ltd.Shanghai 200940China . 宝钢技术研究：英文版 . 2010,第S1期
• 5. 螺杆泵专用抽油杆标准扭矩值的确定方法 [C] . 孙良伟 . 第十四届石油工业标准化学术论坛 . 2013
• 6. 地铁车辆制动模块疲劳强度研究 [A] . 陈姝枚 . 2017