method for preparing a dehydrogeneringskatalysator and method of dehydrogeneren of an alkene to a normal alkene under normal application of this catalyst.

• 1. 由烷烃的脱氢生产烯烃的催化剂和使用所述催化剂生产烯烃的方法 [P] . 中国专利： CN111295243A . 2020-06-16
• 2. 用于正丁烷直接脱氢的铂/锡/氧化铝催化剂的制备方法以及使用所述催化剂制备C4 烯烃的方法 [P] . 中国专利： CN104096561A . 2014-10-15
• 3. method for the oxidation of olefins to unsaturated aldehydes and acids.the oxydatief dehydrogeneren of alkenes to alkadienen and ammoxyderen of unsaturated alkenes to nitrilen in presence of antimoonkatalysatoren. [P] . NL156123B . 1978-03-15

  机译： 烯烃氧化为不饱和醛和酸的方法

• 4. method for dehydrogeneren of alkene, a cycloalkaan, a monohalogeen substituted alkene or cycloalkaan, alkyl substituted aromatic hydrocarbons or mixtures thereof. [P] . NL158154B . 1978-10-16

  机译： 烯烃，环烷烃，单卤代取代的烯烃或环烷烃，烷基取代的芳烃或其混合物的脱氢方法。

• 5. A method for the polymerization of under normal conditions gaseous n - alkenes [P] . 德国专利： DE000001420763C . 1973-05-10

  机译： 正常条件下气态正构烯烃聚合的方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 过程参数对超临界相C10-C14混合正构烷烃催化脱氢制取直链单烯烃反应的影响 [C] . 魏伟 ,孙予罕 ,钟炳 . 第三届全国超临界流体技术学术及应用研讨会 . 2000
• 3. 丙烷氧化脱氢制烯烃Li基催化剂的研究 [A] . 吴一敏 . 2017