APPARATUS FOR DETECTING AND SEPARATING ORGANIC SOLVENT CONTAINED IN WASHING EXHAUST WATER

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  机译： 检测和分离污水中有机溶剂的分离装置

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  机译： 用于检测有机溶剂中水滴的组合物，在有机溶剂中检测水滴的装置和检测有机溶剂中的水滴的方法

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  机译： 用于检测有机溶剂中水滴的组合物，在有机溶剂中检测水滴的装置和检测有机溶剂中的水滴的方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Slag-washing water of blast furnace power station with supercritical organic Rankine cycle [J] . 肖松 ,吴淑英 ,郑东升 . 中南大学学报 . 2013,第003期
• 3. Energy and exergy recovery from exhaust hot water using organic Rankine cycle and a retrofitted configuration [J] . 孙文强 ,岳晓宇 ,王彦辉 . 中南大学学报 . 2018,第06)期
• 4. 分离装置中导爆索与削弱槽对中度射线检测技术 [C] . 刘伟 ,黄绍宇 ,白冰 . 2017航天先进制造技术国际研讨会 . 2017
• 5. 金黄色葡萄球菌凝集素磁分离和快速检测方法的建立及10mL体系磁分离装置的设计 [A] . 杨国泰 . 2019