STEPLESS CONTROL OF U235 TAIL CONCENTRATION TO GAS CENTRIFUGAL SEPARATOR CASCADE FOR URANIUM ISOTOPE SEPARATIONAND CENTRIFUGAL SEPARATOR CASCADE

• 1. 控制离心分离器的方法以及离心分离器 [P] . 中国专利： CN113164981A . 2021-07-23
• 2. 控制离心分离器的方法和离心分离器 [P] . 中国专利： CN113646091A . 2021-11-12
• 3. STEPLESS CONTROL OF U235 TAIL CONCENTRATION TO GAS CENTRIFUGAL SEPARATOR CASCADE FOR URANIUM ISOTOPE SEPARATIONAND CENTRIFUGAL SEPARATOR CASCADE [P] . 日本专利： JPH0256131B2 . 1990-11-29

  机译： U235尾气浓度对铀同位素分离和离心分离器级联的气体离心分离罐的无级控制

• 4. METHOD AND APPARATUS FOR THE INFINITELY VARIABLE SETTING OF THE TAILS U235 CONCENTRATION OF GAS CENTRIFUGE CASCADES FOR THE SEPARATION OF URANIUM ISOTOPES [P] . CA1242677A . 1988-10-04

  机译： 用于终极铀同位素分离的尾气U235浓度无级设置的方法和装置

• 5. METHOD AND APPARATUS FOR THE INFINITELY VARIABLE SETTING OF THE TAILS OF U235 CONCENTRATION OF GAS CENTRIFUGE CASCADES FOR THE SEPARATION OF URANIUMM ISOTOPES [P] . 英国专利： GB2144059B . 1986-04-30

  机译： 铀浓缩同位素分离的U235浓缩尾气的尾部无穷可变设置的方法和装置

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Biogas Production from Sheep Manure by a Simulated Underground Burial System Heated with Cascade-Controlled Solar Water Heated System, as an Indicator of Biomass Potential Contribution to Power Mix in Jordan [J] . Taha M. Alkhamis ,Ali I. Alzoubi ,Bahieh M. Alma’atah . 环境保护（英文） . 2021,第2期
• 3. 离散流量离心分离级联的数值优化 [C] . 彦建宇 ,曾实 ,姜东君 . 第十届全国“三核”论坛暨江西省核学术年会 . 2013
• 4. 离心分离--电催化炭膜耦合处理含油废水的研究 [A] . 李玉霞 . 2019