METHOD OF DECREASING OF CONTENT OF 3-METHYL-4-NITROPHENOL IN 0,0-DIMETHYL-0-/3-METHYL-4-NITROPHENYL/TIOPHOSPHATE

• 1. 借助电化学法制备(4S)-4-(4-氰基-2-甲氧基苯基)-5-乙氧基-2,8-二甲基-1,4-二氢-1,6-萘啶-3-甲酰胺和回收(4S)-4-(4-氰基-2-甲氧基苯基)-5-乙氧基-2,8-二甲基-1,4-二氢-1,6-萘啶-3-甲酰胺的方法 [P] . 中国专利： CN108137587A . 2018-06-08
• 2. 3-三氟甲基-4-硝基苯酚在灭杀或防治福寿螺中的应用、灭杀或防治的方法和药剂 [P] . 中国专利： CN113951255A . 2022-01-21
• 3. METHOD OF 0,0-DIMETHYL-0-/3-METHYL-4-NITROPHENYL/PHOSPOHOROTHIOATE PRODUCTION [P] . CS267193B1 . 1990-02-12

  机译： 生产0,0-二甲基-0- / 3-甲基-4-硝基苯基/磷酸根的方法

• 4. METHOD OF 0,0-DIMETHYL-0-/3-METHYL-4-NITROPHENYL/PHOSPOHOROTHIOATE PRODUCTION [P] . CS8706735A1 . 1989-06-13

  机译： 生产0,0-二甲基-0- / 3-甲基-4-硝基苯基/磷酸根的方法

• 5. METHOD OF 0,0-DIMETHYL-0-/3-METHYL-4-NITROPHENYL/PHOSPOHOROTHIOATE PRODUCTION [P] . CS673587A1 . 1989-06-13

  机译： 生产0,0-二甲基-0- / 3-甲基-4-硝基苯基/磷酸根的方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Improved methods for decreasing stresses of concrete slab of large-span through tied-arch composite bridge [J] . 周德 ,叶梅新 ,罗如登 . 中南大学学报 . 2010,第003期
• 3. 利用DSC数据推测3-甲基4-硝基苯酚的绝热至爆时间 [C] . 石未凡 ,陈鏖 ,曾洪字 . 中国化学会第三届全国热分析动力学与热动力学学术会议暨江苏省第三届热分析技术研讨会 . 2011
• 4. 7β-苯乙酰氨基-3-[4-(1-甲基-4-吡啶鎓)-2-噻唑巯基)]-3-头孢烯-4-羧酸二苯甲基酯碘化物的合成 [A] . 刘鹏 . 2017