Data backup device processing microcomputer compatible with the IBM PC XT / at.

• 1. 一种使用IBM PC XT电脑的可视图文终端设备 [P] . 中国专利： CN1122479A . 1996-05-15
• 2. PCIe设备的兼容处理方法、装置、设备及存储介质 [P] . 中国专利： CN111581139A . 2020-08-25
• 3. the interface of control irrigation can drop a drop in a computer compatible "ibm pcxt or at. [P] . MA23580A1 . 1995-12-31

  机译： 控制灌溉的界面可以在兼容计算机的“ ibm pcxt”或“ at”计算机上滴下。

• 4. DISK DATA ACCESS CONTROLLER ON DISK DRIVES FOR IBMPC/XT/AT COMPATIBLE MICROCOMPUTERS [P] . 英国专利： GB9209494D0 . 1992-06-17

  机译： IBMPC / XT / AT兼容微型计算机的磁盘驱动器上的磁盘数据访问控制器

• 5. High-speed transfer of data between a PC compatible microcomputer and a bus device [P] . 美国专利： US5680553A . 1997-10-21

  机译： 在PC兼容的微型计算机和总线设备之间进行高速数据传输

• 1. THE METHOD OF DATA PROCESSING OF THE ELECTRICAL SURVEYING AND THE PROGRAM SYSTEM USED ON MICROCOMPUTER [J] . 李志聃, 高绋麟 中国矿业大学学报：英文版 . 1990,第001期
• 2. Heart rate variability system based on microcomputer IBM PC/XT [C] . Tkacz E., Antosz P. Engineering in Medicine and Biology Society, 1989. Images of the Twenty-First Century., Proceedings of the Annual International Conference of the IEEE Engineering in . 1989
• 3. Investigation on Inter-layer Dielectric Processed by Chemical Mechanical Polishing and Spin-on Dielectric for Complementary Metal-oxide-semiconductor Compatible Devices [D] . Saman, Rahimah Mohd. 2016
• 4. A collection of programs for nucleic acid and protein analysis written in FORTRAN 77 for IBM-PC compatible microcomputers. [O] . B F Lang, G Burger 1986
• 5. GS MRDS : a system based on the data fields used in the national MRDS system but using dBASE III and a microcomputer (IBM PC) or compatible) for organizing data on mineral resource occurrences and providing tabular and graphic output [O] . Richard B. Taylor, Gary I. Selner, Bruce R. Johnson 1986
• 6. Implementation of a General Finite Element Code on an IBM PC Compatible Microcomputer [R] . Ruesch, R. E. 1984

• 1. IBM PC／XT与IBM PC／XT双机通信系统的设计与实现 [J] . 肖德宝 . 通信与计算机 . 1990,第001期
• 2. An Algorithm and Data Process Scheme for Indoor Location Based on Mobile Devices [J] . Bin Wen ,Ruoshan Kong . 电脑和通信（英文） . 2014,第2期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. Ultra-Fast Next Generation Human Genome Sequencing Data Processing Using DRAGENsupTM/supBio-IT Processor for Precision Medicine [J] . Amit Goyal ,Hyuk Jung Kwon ,Kichan Lee . 遗传学期刊（英文） . 2017,第1期
• 5. 用IBM—PC/XT微型计算机对直流电桥检定进行数据处理 [C] . 方钧烈 . 第三次全国电磁计量测试技术学术会 . 1986
• 6. 微型计算机开关电源PCB的电磁兼容仿真分析 [A] . 裘霄楠 . 2007