A METHOD OF INCREASING THE MELTBLOWN JET THERMAL CORE LENGTH VIA HOT AIR ENTRAINMENT.

• 1. 一种无旁路引风船舶废气脱硫系统及其引风方法 [P] . 中国专利： CN103990355B . 2016.03.02
• 2. 一种无旁路引风船舶废气脱硫系统及其引风方法 [P] . 中国专利： CN103990355A . 2014-08-20
• 3. A METHOD OF INCREASING THE MELTBLOWN JET THERMAL CORE LENGTH VIA HOT AIR ENTRAINMENT [P] . 欧洲知识产权局专利： EP1348049B1 . 2009-11-25

  机译： 通过引热来增加熔喷热芯长度的方法。

• 4. Method of increasing the meltblown jet thermal core length via hot air entrainment [P] . 美国专利： US6613268B2 . 2003-09-02

  机译： 通过夹带热空气增加熔喷射流热芯长度的方法

• 5. A METHOD OF INCREASING THE MELTBLOWN JET THERMAL CORE LENGTH VIA HOT AIR ENTRAINMENT [P] . 欧洲知识产权局专利： EP1348049A1 . 2003-10-01

  机译： 通过引热来增加熔喷热芯长度的方法。

• 1. A METHOD OF ADJUSTING BAR-LENGTH OF STATICALLY INDETERMINATE TRUSS FOR INCREASING ITS LOAD CAPACITY [J] . 隋允康, 邵建义 应用数学和力学（英文版） . 2002,第003期
• 2. Results of Subcritical One- Phase Coaxial Jet Spread Angles and Subcritical to Supercritical Acousticall- Forced Coaxial Jet Dark Core Lengths [C] . J. Rodriguez, I. Leyva, B. Chehroudi, AIAA/ASME/SAE/ASEE Joint Propulsion Conference Exhibit . 2008
• 3. A thermally wavelength-tunable photonic switch based on silicon microring resonator. [D] . Wang, Xuan. 2009
• 4. Nd3+ sensitized core-shell-shell nanocomposites loaded with IR806 dye for photothermal therapy and up-conversion luminescence imaging by a single wavelength NIR light irradiation [O] . Syue-Liang Lin, Zi-Rong Chen, C. Allen Chang 2018
• 5. Phosphorylation of a central clock transcription factor is required for thermal but not photic entrainment. [O] . Euna Lee, Eun Hee Jeong, Hyun-Jeong Jeong, 2014
• 6. Results on Subcritical One-Phase Coaxial Jet Spread Angles and Subcritical to Supercritical Acoustically Forced Coaxial Jet Dark Core Lengths (Preprint); Conference paper [R] . Talley, D., Rodriguez, J. I., Leyva, I. A., 2008

• 1. Increasing the Energy Efficiency of Air Jet Weaving Based on a Novel Method to Exploit Energy Savings Potentials in Production Processes of the Textile Industry [J] . Corrado Grassi1 ,Achim Schroter2 ,Yves-Simon Gloy2 . 环境科学与工程：B . 2016,第001期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. New approaches to increase intestinal length: Methods used for intestinal regeneration and bioengineering [J] . Ali Shirafkan ,Mauro Montalbano ,Joshua Mc Guire . 世界移植杂志 . 2016,第1期
• 4. Transmission spectrum method to measure the photonic bandgap of hollow-core photonic crystal fiber [J] . WEI Hong-yan HOU Lan-tian ZHAO Xing-tao LIU Zhao-lun WANG Wei MA Jing-rui . 光电子快报：英文版 . 2007,第1期
• 5. 增加深水安全作业周期的高黏段塞长度设计方法 [C] . CHEN Chun-lei ,陈春雷 ,WANG Zhi-yuan . 第十三届全国水动力学学术会议暨第二十六届全国水动力学研讨会 . 2014
• 6. 皮芯型聚酯/聚烯烃纺粘熔喷复合成型及产品性能的研究 [A] . 李亚兵 . 2015