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Apparatus and method for separating dust constituents out of an explosive dust-air mixture

机译：从爆炸性粉尘-空气混合物中分离出粉尘成分的设备和方法

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摘要

A method and an apparatus (10) for separating dust constituents out of an explosive dust-air mixture which is passed through a filter housing (26) with filter elements having filter surfaces use a dust container (64), which can be connected in a gastight manner to the filter housing (26), contains inert additive particles (60), into which the process dust produced as a result of the filter surfaces being cleaned down drops and from which the additive particles (60) can be blown onto the filter surfaces by means of compressed air (138). There is a weighing device (122, 124) for the dust container (64).

机译：用于从爆炸性粉尘-空气混合物中分离出灰尘成分的方法和设备（ 10 ），该方法和设备通过带有过滤器元件的带有过滤器表面的过滤器壳体（ 26 ）使用集尘容器（ 64 ），该集尘容器可以气密地连接到过滤器外壳（ 26 ），并包含惰性添加剂颗粒（ 60 >），由于清洁了过滤器表面而产生的工艺粉尘会滴落到其中，并且可以通过压缩空气（ B> 60 ）从其中吹出添加剂颗粒（ 60 ） B> 138 ）。灰尘容器（ 64 ）有一个称重设备（ 122，124 ）。 展开▼

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公开/公告号US7351273B2

专利类型

公开/公告日2008-04-01

原文格式PDF

申请/专利权人 SIEGFRIED HANDTE;
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申请/专利号US20040978213

发明设计人 SIEGFRIED HANDTE;
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申请日2004-10-28

分类号B01D46/46;B01D46;

国家 US

入库时间 2022-08-21 20:09:46

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2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

3. 高炉污泥处理用的水力旋流设备——从高炉粉尘中分离富锌分的方法 [C] . Stein Callenfels JE van . 2003中国钢铁年会 . 2003

4. 工业设备抗粉尘爆炸性能研究 [A] . 李刚 . 2001