method of conditioning variable feedback of signals and system for acquisition, spectral analysis and digital management of data

• 1. 焊接电源反馈信号的生成装置、调节方法及其反馈功率输出信号反馈控制装置 [P] . 中国专利： CN101528404B . 2012.05.16
• 2. 一种异常信号显示方法及频谱分析系统、数字频谱分析仪 [P] . 中国专利： CN108627695B . 2020.06.16
• 3. Analog-to-digital conversion circuit and method for converting analog signals to digital signals in data acquisition systems [P] . 韩国专利： KR100276784B1 . 2001-01-15

  机译： 在数据采集系统中将模拟信号转换为数字信号的模数转换电路和方法

• 4. DIGITAL CONTENTS REPRODUCTION DEVICE, DATA ACQUISITION SYSTEM, DIGITAL CONTENTS REPRODUCTION METHOD, METADATA MANAGEMENT METHOD, ELECTRONIC WATERMARK EMBEDDING METHOD, PROGRAM, AND RECORDING MEDIUM [P] . 日本专利： JP2002351878A . 2002-12-06

  机译： 数字内容复制设备，数据获取系统，数字内容复制方法，元数据管理方法，电子水印嵌入方法，程序和记录介质

• 5. Systems and methods for real-time data quantification, acquisition, analysis, and feedback [P] . US10973439B2 . 2021-04-13

  机译： 用于实时数据量化，采集，分析和反馈的系统和方法

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• 5. Extending the virtual refrigerant charge sensor (VRC) for variable refrigerant flow (VRF) air conditioning system using data-based analysis methods [J] . Li Guannan, Hu Yunpeng, Chen Huanxin, Applied thermal engineering: Design, processes, equipment, economics . 2016,第Null期
• 6. Data analysis methods for a small field-of-view combined scintimammography/digital X-ray system in breast lesion management [J] . D. Kieper, T.D. Green, R. Hoefer, Nuclear Instruments & Methods in Physics Research. Section A, Accelerators, Spectrometers, Detectors and Associated Equipment . 2003,第1期
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• 9. Methodologies for Medical Computing. Date Bases and Management Database Management: Coordinating a Multicenter Trial Using a Microprocessor Based Clinical Research Data Acquisition System [O] . Kathleen F. Swisher, Henry Moore, Yuko Y. Palesch 1983
• 10. Volume 2, Issue 3, Special issue on Recent Advances in Engineering Systems (Published Papers) Articles Transmit / Received Beamforming for Frequency Diverse Array with Symmetrical frequency offsets Shaddrack Yaw Nusenu Adv. Sci. Technol. Eng. Syst. J. 2(3), 1-6 (2017); View Description Detailed Analysis of Amplitude and Slope Diffraction Coefficients for knife-edge structure in S-UTD-CH Model Eray Arik, Mehmet Baris Tabakcioglu Adv. Sci. Technol. Eng. Syst. J. 2(3), 7-11 (2017); View Description Applications of Case Based Organizational Memory Supported by the PAbMM Architecture Martín, María de los Ángeles, Diván, Mario José Adv. Sci. Technol. Eng. Syst. J. 2(3), 12-23 (2017); View Description Low Probability of Interception Beampattern Using Frequency Diverse Array Antenna Shaddrack Yaw Nusenu Adv. Sci. Technol. Eng. Syst. 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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Design of Power Communication Data Network Management System and Implementation of Data Acquisition Module [J] . Yue Zhang ,Zefeng Jian ,Hao Wang . 电子研究与应用 . 2021,第6期
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• 4. 基于心电信号和体动信号的睡眠环境反馈调节系统研究 [A] . 李念 . 2020