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MRI THERMAL IMAGING OF WATER TISSUE AND FAT TISSUE USING TRANSVERSE RELAXOMETRY DATA AND PROTON RESONANCE FREQUENCY SHIFT DATA

机译：横向横切面数据和质子共振频率偏移数据对水组织和脂肪组织的MRI热成像

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摘要

An apparatus (300, 400, 500) comprising a magnetic resonance imaging system (302), the magnetic resonance imaging system comprising: a magnet (306) adapted for generating a magnetic field for orientating the magnetic spins of nuclei of a subject (310) located within an imaging volume (308); a radio frequency transceiver (320) adapted for acquiring magnetic resonance data (346) using a radio frequency coil (318); a computer system (336) comprising a processor (338), wherein the computer system is adapted for controlling the apparatus; and a memory (342, 344) containing machine readable instructions (354, 356, 358, 360, 362), wherein execution of the instructions cause the processor to perform the steps of: acquiring (100, 204) magnetic resonance data using the magnetic resonance imaging system, wherein the magnetic resonance data comprises transverse relaxometry data, and calculating (102, 206) the temperature of the subject within a temperature measurement volume (332) in accordance with the transverse relaxometry data.

机译：一种包括磁共振成像系统（ 302 ）的设备（ 300、400、500 ），该磁共振成像系统包括：磁体（ 306 ）适于产生磁场，该磁场用于定向位于成像空间（ 308 ）内的对象（ 310 ）的原子核的磁自旋;适于使用射频线圈（ 318 ）获取磁共振数据（ 346 ）的射频收发器（ 320 ）;包括处理器（ 338 ）的计算机系统（ 336 ），其中，该计算机系统适于控制该设备;存储器（ 342、344 ）包含机器可读指令（ 354、356、358、360、362 ），其中指令的执行使处理器执行以下步骤使用磁共振成像系统获取（ 100，204 ）磁共振数据，其中磁共振数据包括横向弛豫法数据，并计算（ 102，206 ）根据横向松弛法数据在温度测量体积（ 332 ）内的对象温度。 展开▼

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公开/公告号US2013158387A1

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公开/公告日2013-06-20

原文格式PDF

申请/专利权人 JUKKA ILMARI TANTTU;
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申请/专利号US201113817912

发明设计人 JUKKA ILMARI TANTTU;
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申请日2011-08-26

分类号A61B5/055;A61B18/24;A61N7/02;A61B18/12;A61B5/01;A61B5;

国家 US

入库时间 2022-08-21 16:51:36

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1. 利用横向弛豫时间测量数据和质子共振频率偏移数据的储水组织和脂肪组织的MRI热成像 [P] . 中国专利： CN103080764B . 2015.11.25

2. 利用横向弛豫时间测量数据和质子共振频率偏移数据的储水组织和脂肪组织的MRI热成像 [P] . 中国专利： CN103080764A . 2013-05-01

3. MRI THERMAL IMAGING OF WATER TISSUE AND FAT TISSUE USING TRANSVERSE RELAXOMETRY DATA AND PROTON RESONANCE FREQUENCY SHIFT DATA [P] . 欧洲知识产权局专利： EP2612161B1 . 2020-03-18

机译：横向横切面数据和质子共振频率偏移数据对水组织和脂肪组织的MRI热成像

4. MRI thermal imaging of water tissue and fat tissue using transverse relaxometry data and proton resonance frequency shift data [P] . 美国专利： US10219722B2 . 2019-03-05

机译：使用横向弛豫数据和质子共振频移数据对水组织和脂肪组织进行MRI热成像

5. MRI THERMAL IMAGING OF WATER TISSUE AND FAT TISSUE USING TRANSVERSE RELAXOMETRY DATA AND PROTON RESONANCE FREQUENCY SHIFT DATA [P] . 欧洲知识产权局专利： EP2612161A1 . 2013-07-10

机译：横向横切面数据和质子共振频率偏移数据对水组织和脂肪组织的MRI热成像

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9. MODERATED EPOSTERS1385Longitudinal strain assessment in dilated cardiomyopathy patients using a novel accelerated DENSE sequence1407Simultaneous T1 and T2 cardiac quantification with CABIRIA: initial clinical experience1423Head-to-head comparison of acceleration algorithms in 4-dimensional flow CMR1502Left ventricular function and size evaluated by hybrid cardiac positron emission tomography-magnetic resonance: Intraindividual comparison of left ventricular ejection fraction and ventricular volumes derived by two modalities1510Left Atrium assessed by Cardiovascular Magnetic Resonance at 1.5 and 3 Tesla – age and gender effects1514Comparison of Free Breathing Cardiac MRI Radial technique to the Standard Multi breath-hold cine SSFP CMR technique for the assessment of LV Volumes and Function1536Self-navigated free-breathing isotropic 3D whole heart phase sensitive inversion recovery magnetic resonance without navigator for detection of myocardial infarction1547Assessment of Right Ventricular Strain Using Myocardial Deformation Recovery Semi Automated Technique: Initial Experience and Normal Values1586Tissue tracking myocardial deformation analysis and prediction of left ventricular remodeling in acute myocardial infarction1589Investigating strategies for optimal 31P MRS clinical cardiac at 3T: Initial Results1620Quantitative Criteria for the Diagnosis of the Congenital Absence of Pericardium by Cardiac Magnetic Resonance1632Widespread tissue injury during acute myocardial infarction: evidence from advanced CMR relaxometry1322Computed tomography coronary angiography verSus sTRess cArdiac magneTic rEsonance for the manaGement of sYmptomatic revascularized patients: a cost effectiveness study (STRATEGY study)1339Comparison of low- versus high-dose of gadobutrol for late gadolinium enhancement imaging at 1.5 Tesla: a clinical feasibility study1347Multi-parametric Cardiac Magnetic Resonance for Prediction of Cardiac Complications in Thalassemia Intermedia: a Prospective Multicenter Study1461Prognostic value of Cardiovascular Magnetic Resonance derived indexes of myocardial fibrosis in heart transplant recipients1523The role of CMR in the acute phase of hospitalization: changing paradigms1542Preoperative CMR-based score predict ventricular response after surgical left ventricular reconstruction in ischemic heart failure patients1555Excellent response rate to cardiac resynchronization therapy guided with magnetic resonance imaging1626The ECG as a predictor of arrhythmogenic substrate on Cardiac Magnetic Resonance Imaging in patients undergoing ablation for premature ventricular contractions1649Comparison of T1-mapping at 3.0T CMR and angiographic APPROACH score for area at risk assessment in ST-segment elevation myocardial infarction1340Pathological correlates of left bundle branch disease in patients with non-ischemic cardiomyopathy: a cardiovascular magnetic resonance study1342Myocardial remodelling and fibrosis in nonischaemic dilated cardiomyopathy: insights from cardiovascular magnetic resonance1411The association between fibrosis and contractile dysfunction in hypertrophic cardiomyopathy assessed by cardiovascular magnetic resonance1622Persistent myocardial inflammation due to intramyocardial haemorrhage in reperfused STEMI as a precursor to adverse LV remodelling - insights from multi-parametric mapping1566Semiquantitative analysis of low and high b value DWI for detecting myocardial edema in acute myocarditis1567Value of Cardiac MRI In Detecting Coronary Artery Disease In Newly Diagnosed Systolic Dysfunction1570Usefulness of cardiac magnetic resonance in tuberous sclerosis complex1578Papillary muscles offer further insight into hypertrophied hearts: a cardiovascular magnetic resonance study1627Diagnostic and clinical implications of CMR timing (early versus late) in patients with troponin positive acute coronary syndromes and unobstructed coronary arteries: Table 1. [O] . Upasana Tayal, Alexandros Kallifatidis, P. Garg, 2016

1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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5. 基于LHC上ATLAS探测器收集的13TeV质子与质子对撞数据在矢量玻色子融合过程中寻找重的中性共振态 [A] . 张广义 . 2017