Composition For Ginsenoside K Mediated Retinal Regeneration in Normal Ageing And Macular Degeneration

摘要

The present invention relates to a composition for delaying, preventing and treating a macular degenerative disease which comprises a ginsenoside compound K (compound K) as an active ingredient. More particularly, the present invention relates to a composition for killing a ginsenoside compound K compound K) as an active ingredient for delaying, preventing and treating macular degeneration diseases. The present invention relates to a composition for preventing and treating deterioration of eye function comprising a ginsenoside compound K (compound K) as an active ingredient, and more particularly to a composition for preventing and treating ocular dysfunction, ) As an active ingredient. Degenerative changes in the transport function of the Bruch's membrane due to aging lead to visual disturbances in the elderly and cause severe macular degeneration (AMD), which eventually leads to blindness. The composition according to the present invention improves transport to remove substances deposited on the bruch membrane and enable regeneration of the bruch membrane through cellular reactions in the retinal pigment epithelium (RPE). This treatment is degenerative as it grows older, making it possible to prevent, prevent and treat functional failure of the eye. Decreased transport of vitamin A, essential metals and antioxidants through the bruch membrane is a cause of the aged visual acuity disorder, which can be prevented by the administration of the composition of the present invention. The composition according to the present invention improves the transport of the aged Bruch's membrane and can prevent the deterioration of visual acuity of the general elderly and macular degeneration patients. The composition according to the present invention is applicable to the general public to maintain visual health, and also has the effect of delaying or treating the degree of disease progression in patients with macular degeneration and those with macular degeneration. The composition according to the present invention removes wastes accumulated on the bruch membrane, separates the unused enzymes which are necessary for the recovery of the membrane but is bound to the membrane, and improves the regeneration ability of the bruch membrane through activation of the RPE.