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WATER DETECTION INSIDE ELEVATOR PIT

机译：电梯坑内的水检测

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摘要

A method of detecting water within a pit of an elevator shaft including: emitting an electromagnetic wave from an electromagnetic wave source towards a bottom of an elevator shaft; detecting a reflected electromagnetic wave of the electromagnetic wave using an electromagnetic wave detector; and determining whether water is present within the pit of the elevator shaft in response to the reflected electromagnetic wave.

机译：一种检测电梯井坑内水的方法，包括：从电磁波源向电梯井底部发射电磁波;使用电磁波检测器检测电磁波的反射电磁波;响应于反射的电磁波，确定在电梯井道的凹坑内是否存在水。

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公开/公告号US2020095091A1

专利类型
公开/公告日2020-03-26

原文格式PDF
申请/专利权人 OTIS ELEVATOR COMPANY;
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申请/专利号US201916578632
发明设计人 ANKIT ANAND GUPTA;STELLA M. OGGIANU;GIRISHA DJ;ARANGARAJAN P;ANUP KUMAR;
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申请日2019-09-23
分类号B66B5;G01V8/12;B66B5/02;B66B1/34;B66B3;G08B7/06;
国家 US
入库时间 2022-08-21 11:22:10

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2. 沃特世荣获Pittcon 2007金奖Waters Synapt HDMS质谱分析系统荣获Pittcon新产品金奖 [J] . . 药物分析杂志 . 2007,第3期

3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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5. 检测电梯导轨垂直度的新方法及其智能检测仪 [C] . 孙立新 ,周蕊 ,公永和 . 2005年特种设备安全国际论坛 . 2005

6. 石塘水阳西湾至罗湖水电梯级开发水环境影响研究 [A] . 刘莹莹 . 2011