...
机译:微生物群通过激活产生IL-10的调节性B细胞中的TLR2/MyD88/PI3K信号传导来维持结肠稳态
UNC, Dept Med, Div Infect Dis, Chapel Hill, NC USA;
Cincinnati Childrens Hosp, Res Ctr, Dept Pediat, Div Mol Immunol, Cincinnati, OH USA;
Univ N Carolina, Div Gastroenterol & Hepatol, Dept Med, Ctr Gastrointestinal Biol & Dis, Room 7309AUNC, Dept Genet, Lineberger Comprehens Canc Ctr, Chapel Hill, NC USAUNC, Dept Microbiol & Immunol, Chapel Hill, NC USAUNC, Dept Nutr, Chapel Hill, NC USA;
机译:CD8(+)CD122(+)CD49d(low) regulatory T cells maintain T-cell homeostasis by killing activated T cells via Fas/FasL-mediated cytotoxicity
机译:SHPS-1 and a synthetic peptide representing its ITIM inhibit the MyD88, but not TRIF, pathway of TLR signaling through activation of SHP and PI3K in THP-1 cells.
机译:Regulation of PTEN translation by PI3K signaling maintains pathway homeostasis
机译:Retraction: Tanshinone IIA prevents left ventricular remodelling via the TLR4/MyD88/NF‐κB signalling pathway in rats with myocardial infarction. Dong‐Mei Wu Yong‐Jian Wang Xin‐Rui Han Xin Wen Lei Li Lan Xu Jun Lu and Yuan‐Lin Zheng. J Cell Mol Med. 2018; 22: 3058–3072 (https://doi.org/10.1111/jcmm.13557).
机译:Cell Entry of avian Reovirus Follows a Caveolin-1-mediated and Dynamin-2-dependent Endocytic pathway That Requires activation of p38 mitogen-activated protein Kinase (mapK) and src signaling pathways as Well as microtubules and small GTpase Rab5 protein