...
机译:Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice
Fujita Hlth Univ, Inst Comprehens Med Sci, Div Therapies Against Intractable Dis, Toyoake, Aichi 4701192, Japan;
Univ Tokushima, Inst Enzyme Res, Tokushima 770, Japan;
Kitasato Univ, Sch Vet Med & Anim Sci, Lab Expt Anim Sci, Towada, Aomori 034, JapanNatl Ctr Neurol & Psychiat, Natl Inst Neurosci, Dept Mol Therapy, Tokyo, JapanUniv Tokushima, Fac Engn, Dept Biol Sci & Technol, Tokushima 770, JapanKawasaki Med Univ, Dept Internal Med, Div Neurol, Okayama, Japan;
Duchenne muscular dystrophy; activin; therapy; myostatin blockade; MUSCULAR-DYSTROPHY; BINDING-PROTEIN; ACTIVIN; GENE; GROWTH; HYPERTROPHY; PROPEPTIDE; RECEPTOR; DIFFERENTIATION; HYPERPLASIA;