Since COVID-19 was declared a pandemic in March 2020, there has been extensive human-to-human transmission, resulting in substantial respiratory pathology and manifestations outside the pulmonary parenchyma 1,2 . Initial observations showed that the hyper-inflammatory response induced by COVID-19 often led to systemic coagulation derangement and coagulopathy, with thrombosis an important factor in the pathology and clinical course of infection 3–5 .
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