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机译:Chronic exposure to polluted urban air aggravates myocardial infarction by impaired cardiac mitochondrial function and dynamics
Univ Buenos Aires, Fac Farm & Bioquim, Dept Ciencias Quim, C1113AAD, Buenos Aires, DF, Argentina|Univ Buenos Aires, CONICET, Inst Bioquim & Med Mol IBIMOL, C1113AAD, Buenos Aires, DF, Argentina;
Univ Buenos Aires, CONICET, Inst Bioquim & Med Mol IBIMOL, C1113AAD, Buenos Aires, DF, Argentina|Univ Buenos Aires, Fac Med, Dept Patol, C1113AAD, Buenos Aires, DF, Argentina;
Univ Buenos Aires, Fac Farm & Bioquim, Dept Ciencias Quim, C1113AAD, Buenos Aires, DF, Argentina|Univ Buenos Aires, CONICET, Inst Bioquim & Med Mol IBIMOL, C1113AAD, Buenos Aires, DF, Argentina|Univ Freiburg, Univ Heart Ctr Freiburg Bad Krozingen, Fac MedUniv Buenos Aires, Fac Farm & Bioquim, Catedra Inmunol, C1113AAD, Buenos Aires, DF, Argentina|Univ Buenos Aires, CONICET, Inst Estudios Inmunidad Humoral IDEHU, C1113AAD, Buenos Aires, DF, ArgentinaUniv Buenos Aires, CONICET, Inst Biol Celular & Neurociencias IBCN, Buenos Aires, DF, ArgentinaUniv Buenos Aires, Fac Farm & Bioquim, Dept Tecnol Farmaceut, C1113AAD, Buenos Aires, DF, ArgentinaUniv Buenos Aires, Fac Med, Dept Patol, C1113AAD, Buenos Aires, DF, ArgentinaUniv Freiburg, Univ Heart Ctr Freiburg Bad Krozingen, Fac Med, Cardiol & Angiol 1, D-79106 Freiburg, Germany;
Air pollution; Myocardial infarction; Redox metabolism; Mitochondria; PGC-1 alpha; OPA-1;