机译:受损cross-presentation CD8α+ CD11c +树突细胞由日本脑炎病毒TLR2 / MyD88信号pathway-dependent方式
College of Veterinary Medicine, Bio-Safety Research Institute, Chonbuk National University, Jeonju;
Department of Pharmacology, School of Medicine, Pusan National University, Busan, South Korea;
Japanese encephalitis virus; CD11c Antigens; Cross-PresentationHistocompatibility Antigens Class Iantigens;
机译:Lack of Herpes Virus Entry Mediator Signals in Thymocytes Impairs Conventional CD8 T Cell Selection and Promotes Memory-like CD8 T Cell Development
机译:Inhibitory effect of miR-125b on hepatitis C virus core protein-induced TLR2/MyD88 signaling in THP-1 cells
机译:在TLR2 / MyD88信号途径依赖性方式中,日本脑炎病毒的CD8α+ CD11C +树突细胞的交叉呈递受损