A female Tibetan terrier aged five years and six months presented 23 months after portosystemic shunt attenuation due to generalised tonic-clonic epileptic seizure activity. Repeat MRI of the brain showed symmetrical T2-weighted hyperintense lesions affecting the cerebral grey matter bilaterally suggestive of cerebral atrophy and polioencephalomalacia. Cerebral atrophy and polioencephalomalacia could develop after successful attenuation of portosystemic shunt surgery.
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