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机译:apoa-i / sr-bi通过HUVECS中的PI3K / AKT信号通路调节S1P / S1PR2介导的炎症
Univ South China Inst Cardiovasc Dis Key Lab Arteriosclerol Hunan Prov 28 W Changsheng Rd Hengyang 421001 Hunan Peoples R China;
Maternal &
Child Hlth Hosp Hubei Prov Dept Pathol Wuhan 430072 Hubei Province Peoples R China;
Univ South China Dept Histol &
Embryol Hengyang 421001 Hunan Peoples R China;
Chinese Acad Med Sci Natl Lab Med Mol Biol Inst Basic Med Sci Sch Basic Med Peking Union Med Coll Beijing 100005 Peoples R China;
Univ South China Inst Cardiovasc Dis Key Lab Arteriosclerol Hunan Prov 28 W Changsheng Rd Hengyang 421001 Hunan Peoples R China;
Univ South China Inst Cardiovasc Dis Key Lab Arteriosclerol Hunan Prov 28 W Changsheng Rd Hengyang 421001 Hunan Peoples R China;
Univ South China Inst Cardiovasc Dis Key Lab Arteriosclerol Hunan Prov 28 W Changsheng Rd Hengyang 421001 Hunan Peoples R China;
Maternal &
Child Hlth Hosp Hubei Prov Dept Pathol Wuhan 430072 Hubei Province Peoples R China;
Univ South China Affiliated Hosp 1 Clin Res Inst Hengyang 421001 Hunan Peoples R China;
Univ South China Inst Cardiovasc Dis Key Lab Arteriosclerol Hunan Prov 28 W Changsheng Rd Hengyang 421001 Hunan Peoples R China;
HDL; S1P/S1PR2; apoA-I/SR-BI; Inflammation;
机译:APOM-S1P通过HUVECS中的PI3K / AKT信号传导途径调节OX-LDL诱导的炎症
机译:Tricin,Njavara中的类黄酮通过调节p38MAPK和PI3K / Akt途径减少hPBMC中的炎症反应,并预防HUVEC中炎症相关的内皮功能障碍
机译:Tricin,来自Njavara的黄酮类化合物通过调节P38MAPK和PI3K / AKT途径来减少HPBMC中的炎症反应,并防止HUVECS中的炎症相关内皮功能障碍
机译:乳铁蛋白诱导维生素B6的合成,通过激活PDXP和PI3K / AKT / ERK1 / 2路径来保护HUVEC功能
机译:从A(2B)腺苷受体到人脐静脉内皮细胞(HUVEC)中细胞外信号调节激酶(ERK1 / 2)的信号通路及其在HUVEC增殖中的作用
机译:穿心莲内酯可通过HUVEC中的PI3K / AKT-eNOS信号传导来防止HG诱导的炎症细胞凋亡迁移和血管生成受损。
机译:乌纳替肽通过抑制JNK / NF-κB信号通路并激活PI3K / AKT / NRF2途径来衰减小鼠巨噬细胞RAW264.7细胞中的LPS诱导的炎症
机译:分子靶向pI3K / akt通路预防激素抵抗前列腺癌的发生