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机译:MSH6通过调节HIF1A加剧缺氧微环境,以促进胶质母细胞瘤多形形的转移
Harbin Med Univ Affiliated Hosp 1 Dept Magnet Resonance Harbin 150001 Heilongjiang Peoples R China;
Harbin Med Univ Affiliated Hosp 1 Dept Abdominal Ultrasonog Harbin Heilongjiang Peoples R China;
Harbin Med Univ Affiliated Hosp 1 Dept Magnet Resonance Harbin 150001 Heilongjiang Peoples R China;
Shanxi Med Univ Hosp 1 Dept Ultrason Imaging Taiyuan Shanxi Peoples R China;
Harbin Med Univ Affiliated Hosp 1 Dept Abdominal Ultrasonog Harbin Heilongjiang Peoples R China;
Harbin Med Univ Affiliated Hosp 1 Dept Pancreat &
Biliary Surg Harbin Heilongjiang Peoples R China;
Harbin Med Univ Affiliated Hosp 1 Dept Magnet Resonance Harbin 150001 Heilongjiang Peoples R China;
glioblastoma multiforme; MSH6; tumor microenvironment; hypoxia; HIF1A;
机译:缺氧敏化剂AK-2123(Sanazole)对多形胶质母细胞瘤的强度调制放射疗法,采用同时综合增强技术
机译:低氧微环境维持胶质母细胞瘤干细胞并促进向癌症干细胞表型的重编程。
机译:P53和miR-210受调节的神经元,一种神经元碱性螺旋环 - 螺旋转录因子在胶质母细胞瘤患者中下调,并用作缺氧微环境下的肿瘤抑制剂
机译:局部曲率分析以区分多形胶质母细胞瘤与孤立性转移
机译:癌细胞与肿瘤微环境之间的相互交流促进乳腺癌转移
机译:缺氧微环境维持胶质母细胞瘤干细胞并促进对癌症干细胞表型的重编程
机译:缺氧微环境维持胶质母细胞瘤干细胞并促进对癌症干细胞表型的重编程
机译:微环境和信号通路调节早期传播,休眠和转移。