机译:锰暴露加剧了帕金森病的MITOPARK小鼠模型中的渐进式电机缺陷和神经变性:与金属神经毒性的基因和环境相互作用的相关性
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Iowa State Univ Dept Biomed Sci Iowa Ctr Adv Neurotoxicol Parkinson Disorders Res Program Ames;
Manganese; Mitochondria; MitoPark; Neuroinflammation; Animal model; TFAM; Dopamine; Parkinson's disease; Gene-environment interaction;
机译:锰暴露加剧了帕金森病的MITOPARK小鼠模型中的渐进式电机缺陷和神经变性:与金属神经毒性的基因和环境相互作用的相关性
机译:槲皮素对线粒体功能障碍和进步多巴胺能神经变性的分子机制依赖于细胞培养和帕金森病的瘤宫转基因小鼠模型
机译:肌肉非编码RNA靶向GDNF在帕金森病的小鼠模型中救援电机缺陷和神经变性
机译:氧化铈纳米颗粒可预防MPTP诱发的帕金森氏病小鼠模型中的多巴胺能神经变性
机译:帕金森病的基因 - 环境相互作用:抑制囊泡单胺转运蛋白,农药暴露和DJ-1功能丧失
机译:锰暴露会加剧帕金森氏病的MitoPark小鼠模型中的进行性运动功能障碍和神经退行性变:金属神经毒性中与基因和环境相互作用的相关性
机译:认知功能障碍发生在帕金森病的mitopark小鼠模型中的运动症状发作之前。