机译:Rab37直接与ATG5相互作用,通过调节ATG5-12-16复杂组件来促进自噬体形成
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
Wuhan Univ Hubei Key Lab Cell Homoeostasis Coll Life Sci Wuhan 430072 Hubei Peoples R China;
机译:RAB37直接与ATG5相互作用,通过调节ATG5-12-16复杂组件来促进自噬体形成
机译:ATG5结构与其互动合作伙伴的结构展示了自噬成熟的见解
机译:ATG5癌症突变和替代MRNA剪接显示了调节ATG12-ATG5-ATG16L1复合组件和自噬的共轭开关
机译:LRRK2直接与DLP1相互作用以调节线粒体的动力学和功能
机译:后期促进复合物与组蛋白修饰蛋白和染色质组装因子相互作用。
机译:RAB37与ATG5直接相互作用并通过调节ATG5-12-16复合物装配促进自噬体形成
机译:RAB37直接与ATG5相互作用,通过调节ATG5-12-16复杂组件来促进自噬体形成