...
机译:PunicalAGIN通过PI3K / AKT途径的上调诱导NRF2 / HO-1表达,并抑制RAW264.7巨噬细胞中的LPS诱导的氧化应激
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
Shanxi Agr Univ Coll Anim Sci &
Vet Med Taigu 030801 Shanxi Peoples R China;
Beijing Agr Univ Coll Anim Sci &
Technol Beijing 102206 Peoples R China;
Beijing Agr Univ Coll Anim Sci &
Technol Beijing 102206 Peoples R China;
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
Beijing Agr Univ Coll Anim Sci &
Technol Beijing 102206 Peoples R China;
China Agr Univ Coll Vet Med CAU BUA TCVM Teaching &
Researching Team Beijing 100193 Peoples R;
机译:Punicalagin通过上调PI3K / AKT途径诱导Nrf2 / HO-1表达,并抑制RAW264.7巨噬细胞中LPS诱导的氧化应激。
机译:乌司他丁通过抑制JNK /NF-κB信号通路和激活PI3K / Akt / Nrf2通路来减轻LPS诱导的小鼠巨噬细胞RAW264.7细胞的炎症
机译:咖啡因羟色胺通过激活Pi3k / Akt / Nrf2途径上调Ho-1表达,从而保护人类角质形成细胞HaCaT细胞免受H _2O_2诱导的氧化应激和细胞凋亡。
机译:M2-A,一种新颖的阿莫那肽类似物,通过降低拓扑异构酶LIa活性诱导HL60细胞凋亡和G2-M逮捕并抑制PI3K / Akt途径。
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:Punicalagin通过上调PI3K / AKT途径诱导Nrf2 / HO-1表达并抑制RAW264.7巨噬细胞中LPS诱导的氧化应激。
机译:乌纳替肽通过抑制JNK / NF-κB信号通路并激活PI3K / AKT / NRF2途径来衰减小鼠巨噬细胞RAW264.7细胞中的LPS诱导的炎症