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机译:暴露于环境烟草烟雾,人E-cadherin C-160a多态性和儿童哮喘
Division of Allergy Immunology and Rheumatology Department of Pediatrics Tungs' Taichung;
Institute of Medicine Chung Shan Medical University Taichung Taiwan;
Institute of Medicine Chung Shan Medical University Taichung Taiwan;
Department of Public Health Chung Shan Medical University No 110 Chien-Kuo N Road Section 1;
Department of Public Health Chung Shan Medical University No 110 Chien-Kuo N Road Section 1;
Department of Public Health Chung Shan Medical University No 110 Chien-Kuo N Road Section 1;
Institute of Medicine Chung Shan Medical University Taichung Taiwan Department of Pediatrics;
Department of Public Health Chung Shan Medical University No 110 Chien-Kuo N Road Section 1;
Department of Public Health Chung Shan Medical University No 110 Chien-Kuo N Road Section 1;
Department of Rehabilitation Jen-Teh Junior College of Medicine Nursing and Management Miaoli;
机译:暴露于环境烟草烟雾,人类E-钙粘蛋白C-160A多态性和儿童哮喘
机译:一项纵向研究表明,IL13基因多态性改变了暴露于烟草烟雾对儿童持续性喘息和哮喘的影响
机译:细胞间粘附分子1(ICAM1)多态性与环境烟草烟雾对儿童哮喘的相互作用
机译:家庭环境烟草烟雾(ETS)暴露和儿童哮喘风险 - 在荟萃分析中降低研究之间的研究之间的技术
机译:围产期发育期间的环境暴露:风险检查的方法包括对环境烟草烟雾暴露进行化学测量,对有机溶剂暴露进行荟萃分析和风险评估。
机译:一项纵向研究表明IL13基因多态性改变了接触烟草烟雾对儿童持续性喘息和哮喘的影响
机译:纵向研究表明,