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机译:chk1 DNA损伤检查点激酶靶向的Cdc25有丝分裂诱导剂
Department of Molecular Biology, The Scripps Research Institute, La Jolla, CA 92037, USA.;
Adenosinetriphosphatase metabolism; CDC2 Protein Kinase metabolism; Cell Cycle Proteins metabolism; Cell Division; DNA Damage; DNA Helicases metabolism; Fungal Proteins metabolism; G2 Phase; Gamma Rays; Genes; Fungal; Mitosis; Models; Biological; Mutation; Nuclear Proteins; Phosphorylation; Phosphotyrosine metabolism; Protein Kinases genetics metabolism; Protein-Tyrosine Kinase genetics metabolism; Recombinant Fusion Proteins metabolism; Research Support; U.S. Gov't; P.H.S.; Schizosaccharomyces cytology genetics metabolism radiation effects; Signal Transduction; Temperature; ras-GRF1;
机译:P53相关DNA损伤修复蛋白检查点激酶1(CHK1)和生长停滞和DNA损伤诱导45α(GADD45A)在人口鳞状细胞癌中的表达及预后意义
机译:DNA损伤诱导的CHK1磷酸化需要检查点激酶1(CHK1)和微微核糖体维持(MCM)复合物之间的相互作用
机译:哺乳动物中Chk1检查点途径的保护:DNA损伤通过Cdc25与Cdk调控的联系
机译:Epstein-Barr病毒蛋白激酶BGLF4将DNA损伤反应和有丝分裂磷酸化信号传导整合以促进病毒复制
机译:CHK1在维护G2 DNA损伤检查点。
机译:DNA损伤诱导的Chk1磷酸化需要Checkpoint激酶1(Chk1)和微染色体维持(MCM)复合体之间的相互作用。
机译:Checkpoint激酶1(Chk1)-short是Chk1的剪接变体和内源性抑制剂,可调节细胞周期和DNA损伤检查点