机译:SARS-COV-2的NSPL蛋白转化关机和免疫逃避的结构基础
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Univ Munich Lab Funct Genome Anal Munich Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Ulm Univ Inst Mol Virol Med Ctr Ulm Germany;
Univ Munich Gene Ctr Munich Dept Biochem Munich Germany;
机译:增强感染性和免疫逃避SARS-COV-2变体的结构基础
机译:B组链球菌β和肺炎球菌Hic蛋白是结构相关的免疫逃避分子,它们以类似方式结合补体抑制剂因子H。
机译:B组链球菌β和肺炎球菌Hic蛋白是结构相关的免疫逃逸分子,以类似的方式结合补体抑制因子H。
机译:由III型效应器HOPZ3的翻译后修饰通过细菌和植物蛋白的乙酰化调节番茄宿主免疫
机译:SOCS蛋白在HIV免疫逃逸中的作用。
机译:SARS-COV-2的NSP1蛋白的翻译关闭和免疫逃避的结构基础
机译:结构基础,用于增强SARS-COV-2变体的感染性和免疫逃逸