机译:低剂量单体(2-乙基己基)邻苯二甲酸酯通过PPARα依赖性PI3K / AKT / NF-KB途径促进卵巢癌发育
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China Scientific Research Department First Affiliated Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China Department ofGastroenterology Second Affiliated Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Gynecological Oncology Zhejiang Cancer Hospital Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Research Unit of Intelligence Classification of Tumor Pathology and Precision Therapy Chinese Academy of Medical Sciences (2019RU042) Hangzhou 310058 Zhejiang China;
Department of Epidemiology Biostatistics and Occupational Health McGill University. Montreal Canada;
Department of Public Health Hangzhou Normal University School of Medicine Hangzhou China Zhejiang Provincial Center for Uterine Cancer Diagnosis and Therapy Research of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Central Laboratory of the Fourth Affiliated Hospital Zhejiang University School of Medicine Yiwu China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China;
Department of Toxicology of School of Public Health and Department of Gynecologic Oncology of Women's Hospital Zhejiang University School of Medicine Hangzhou China Research Unit of Intelligence Classification of Tumor Pathology and Precision Therapy Chinese Academy of Medical Sciences (2019RU042) Hangzhou 310058 Zhejiang China;
MEHP; Low-dose exposure; Ovarian cancer; Metastasis; PI3K pathway; PPARα;
机译:Cemip通过PI3K / AKT信号通路促进卵巢癌开发和进展
机译:蜗牛在前列腺癌进展过程中通过PI3K / AKT依赖性Rac1激活以及PI3K / AKT依赖性途径促进细胞迁移
机译:蜗牛通过PI3K / AKT依赖的RAC1激活以及在前列腺癌进展期间的PI3K / AKT无关的途径促进细胞迁移
机译:对Mono(2-乙基己基)邻苯二甲酸酯,DI(2-乙基己基)邻苯二甲酸代谢物,大鼠等离子体,羊水和胎儿的邻苯二甲酸酯的开发和验证
机译:结合基因组学和遗传学方法分析人卵巢癌中的PI3K / AKT途径。
机译:蜗牛在前列腺癌进展过程中通过PI3K / AKT依赖性Rac1激活以及PI3K / AKT依赖性途径促进细胞迁移
机译:邻苯二甲酸酯的发育暴露于二(2-乙基己基)通过PI3K / AKT信号通路诱导小脑颗粒细胞凋亡
机译:关于CD-1-swiss小鼠中单(2-乙基己基)邻苯二甲酸酯(CasNO.4376-20-9)的发育毒性的最终报告。第2卷。实验室补充