机译:STIM1和钙调蛋白与Orai1相互作用以诱导Ca〜(2+)依赖性CRAC通道失活
Departments of Pathology, Stanford University School of Medicine, Stanford, CA 94305 Departments of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA 94305;
Departments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305;
Departments of Neurobiology, Stanford University School of Medicine, Stanford, CA 94305;
Departments of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA 94305;
calcium; ion channel gating; store-operated calcium entry; patch-clamp; calcium-binding proteins;
机译:Orai1突变改变CRAC通道的离子渗透和Ca〜(2+)依赖快速灭活:渗透和门控耦合的证据。
机译:校正:Orai1孔残基独立于钙调蛋白控制CRAC通道失活
机译:Orai1孔残基独立于钙调蛋白控制CRAC通道失活
机译:活化的Ca〜(2 +)/钙调蛋白依赖性蛋白激酶II的定位在脊柱内:建模和计算机仿真
机译:当钡(2+)是电荷载体时,L型钙通道的二价离子依赖性失活,并且孔谷氨酸对带电DHP相互作用的不对称贡献。
机译:STIM1和钙调蛋白与Orai1相互作用以诱导CRAC通道的Ca2 +依赖性失活
机译:STIM1和钙调蛋白与Orai1相互作用以诱导CRAC通道的Ca2 +依赖性失活