机译:可溶性环氧化物水解酶缺乏症可改变胰岛的大小,并改善胰岛素抵抗模型中的葡萄糖稳态
Departments of Entomology, University of California, Davis, CA 95616;
Nutrition, University of California, Davis, CA 95616;
Nutrition, University of California, Davis, CA 95616;
Departments of Entomology, University of California, Davis, CA 95616;
Departments of Entomology, University of California, Davis, CA 95616;
Department of Internal Medicine, Division of Nephrology, School of Medicine, University of California, Davis, CA 95616;
Departments of Entomology, University of California, Davis, CA 95616;
Department of Pharmacology and Toxicology, Cardiovascular Research Center, Medical College of Wisconsin, Milwaukee, WI 53226;
Nutrition, University of California, Davis, CA 95616;
Departments of Entomology, University of California, Davis, CA 95616,Cancer Center, University of California, Davis, CA 95616;
type 2 diabetes; pancreas; arachidonic acid pathway;
机译:在链脲佐菌素诱导的糖尿病小鼠模型中抑制可溶性环氧化物水解酶对葡萄糖稳态和胰岛损伤的有益作用
机译:生物素缺乏症对胰岛形态,胰岛素敏感性和葡萄糖稳态的影响。
机译:Alx3缺乏症小鼠的β细胞胰岛素减少,葡萄糖稳态改变和胰岛细胞凋亡增加
机译:亚砷酸盐及其代谢物,甲基胂岩,抑制胰岛胰岛血糖胰岛素分泌期间的钙流量
机译:可溶性环氧化物水解酶抑制作用可减弱血管重塑,并能预防脑缺血。
机译:可溶性环氧化物水解酶缺乏症可改变胰岛的大小并改善胰岛素抵抗模型中的葡萄糖稳态
机译:可溶性环氧化物水解酶缺乏症可改变胰岛大小,并改善胰岛素抵抗模型中的葡萄糖稳态