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机译:烟酰胺可预防NAD + sup>消耗并保护神经元免于兴奋性毒性和脑缺血:SIRT1消耗NAD + sup>可能会危及精力充沛的神经元
Laboratory of Neurosciences National Institute on Aging Intramural Research Program Baltimore MD 21224 USA;
Laboratory of Neurosciences National Institute on Aging Intramural Research Program Baltimore MD 21224 USA;
Laboratory of Neurosciences National Institute on Aging Intramural Research Program Baltimore MD 21224 USA;
Laboratory of Neurosciences National Institute on Aging Intramural Research Program Baltimore MD 21224 USA;
Laboratory of Neurosciences National Institute on Aging Intramural Research Program Baltimore MD 21224 USA;
Excitotoxicity; Glutamate; NMDA; NAD+; NADH; SIRT1; PARP-1; PAR; Nicotinamide; MCAO; TUNEL;
机译:烟酰胺可防止NAD +消耗并保护神经元免受兴奋性毒性和脑缺血:SIRT1消耗NAD +可能会危及能量受损的神经元。
机译:NAD +和烟酰胺:脑缺血中的性别差异
机译:臭氧暴露在脊髓神经元中通过PARP1激活诱导代谢紊乱和NAD +耗尽
机译:SIRT1保护皮质神经元免受剪切应力引起的机械损伤
机译:Nampt介导的NAD +生物合成在海马神经干细胞和兴奋性神经元中的作用。
机译:烟酰胺可防止NAD +耗竭和保护神经元免受兴奋毒性和脑缺血:NAD + SIRT1消耗量可以危及能量损害神经元
机译:烟酰胺可防止NAD +耗竭和保护神经元免受兴奋毒性和脑缺血:NAD + SIRT1消耗量可以危及能量损害神经元