机译:通过受体酪氨酸激酶/ PI3K / AKT信号轴控制星形细胞瘤细胞的增殖,以及使用PI-103和TCN作为潜在的抗星形细胞瘤疗法
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD Molecular Biology and Microbiology,George Mason University, Fairfax, VA;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute-Frederick, Frederick,MD;
akt; astrocytoma; egfr; nfl; p53.;
机译:piplampsia胎盘中受体酪氨酸激酶样孤儿橙子1的下调抑制了Pi3k / akt / mtor途径住宿的人滋养细胞增殖,迁移和侵袭
机译:PI3K / Akt / mTOR信号通路抑制对乳腺癌细胞受体酪氨酸激酶表达的影响
机译:具有高发育能力的卵母细胞周围卵母细胞表现出磷酸肌醇的缩小调节1,3激酶/蛋白激酶B(PI3K / AKT)信号传导基因参与增殖和生存
机译:表皮生长因子受体酪氨酸激酶抑制剂ZD1839(Iressa)通过独立于p53和MMP,uPAR依赖性机制抑制人口腔鳞状细胞癌细胞的增殖和侵袭
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:通过受体酪氨酸激酶/ PI3K / AKT信号轴控制星形细胞瘤细胞的增殖以及使用PI-103和TCN作为潜在的抗星形细胞瘤疗法
机译:通过受体酪氨酸激酶/ PI3K / AKT信号轴控制星形细胞瘤细胞的增殖,以及使用PI-103和TCN作为潜在的抗星形细胞瘤疗法
机译:来自新型受体酪氨酸激酶的信号传导和多能乳腺祖细胞的控制