机译:通过增加21号染色体上DSCR1和DYRK1A的剂量而导致NFAT失调
Stanford Univ, Sch Med, Dept Pathol, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Program Immunol, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Med, Div Cardiovasc Med, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Dev Biol, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Genet, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Howard Hughes Med Inst, Stanford, CA 94305 USA;
Kyoto Univ, Grad Sch Med, HMRO, Genet Engn & Funct Genom Unit, Kyoto 6068501, Japan;
DOWN-SYNDROME PHENOTYPES; TRANSCRIPTION FACTOR; REGULATORY NETWORK; CRITICAL REGION; MOUSE MODEL; CALCINEURIN; MICE; CALCIUM; SIGNALS; ATC;
机译:21号染色体上DSCR1和DYRK1A剂量的增加使NFAT调节不稳定并解释了唐氏综合症的表型
机译:21号染色体上DSCR1和DYRK1A剂量的增加使NFAT调节不稳定并解释了唐氏综合症的表型
机译:DYRK1A和DSCR1剂量的增加会延迟新皮层祖细胞的神经元分化
机译:黑孜然种子油治疗的影响为1.5毫升/天和3毫升/天至白细胞介素-21(IL-21)表达患者的代谢综合征风险
机译:人类21号染色体alpha21-II区的组织和组成。
机译:DYRK1A和DSCR1剂量的增加会延迟新皮层祖细胞的神经元分化
机译:21号染色体上DSCR1和DYRK1A剂量的增加破坏了NFAT的调节作用,并解释了唐氏综合症的表型