机译:过度活跃的Ras引发的白血病对MEK抑制的反应和抵抗
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Mouse Cancer Genetics Program, National Cancer Institute, Fredrick, Maryland 21702, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Medicine, University of California, San Francisco, California 94143, USA;
Department of Anatomy, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Pfizer Global Research and Development, Ann Arbor, Michigan 48105, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
Department of Laboratory Medicine, University of California, San Francisco, California 94143, USA;
Department of Anatomy, University of California, San Francisco, California 94143, USA;
Institute of Molecular and Cell Biology, Singapore 138673, Singapore;
Institute of Molecular and Cell Biology, Singapore 138673, Singapore;
University of Texas Southwestern, Dallas, Texas 75235, USA;
Laboratory of Cellular Oncology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA;
Pfizer Global Research and Development, Ann Arbor, Michigan 48105, USA;
Department of Pediatrics, University of California, San Francisco, California 94143, USA;
机译:替代的替代性促进途径的矛盾激活决定了慢性淋巴细胞白血病中MEK MEK MEK抑制剂的抵抗力
机译:AT13387对HSP90的抑制作用会延迟对BRAF抑制剂的耐药性出现,并克服黑色素瘤模型对BRAF和MEK双重抑制的耐药性
机译:获得的对BRAF抑制的抗性可以赋予对BRAF / MEK组合抑制的交叉耐药性
机译:通过抑制MEK / ERK途径使大肠癌细胞对紫杉醇诱导的细胞凋亡的敏感性取决于caspase-4
机译:注意缺陷多动症(ADHD)中反应抑制的神经基质。
机译:响应和耐mEK抑制作用白血病由活动过度的Ras引发
机译:AT13387对HSP90的抑制延迟了对BRAF抑制剂的抗性的出现,克服了黑色素瘤模型中的双BRAF和MEK抑制