机译:R-spondin 1和Slit2诱导肠道干细胞增强了放化疗保护
Department of Biologic and Materials Sciences, University of Michigan School of Dentistry, Ann Arbor, Michigan 48109, USA;
Department of Biologic and Materials Sciences, University of Michigan School of Dentistry, Ann Arbor, Michigan 48109, USA;
Departments of Cell and Developmental Biology and Internal Medicine,Division of Gastroenterology, University of Michigan School of Medicine, Ann Arbor, Michigan 48109, USA;
Department of Biologic and Materials Sciences, University of Michigan School of Dentistry, Ann Arbor, Michigan 48109, USA;
机译:Wnt和R-spondin配体在Lgr5(+)肠干细胞自我更新过程中的非等效性
机译:Slit2 / Robo1通过激活肠道干细胞的自噬来减轻DSS诱导的溃疡性结肠炎
机译:树突状细胞对TGFβ2的激活驱动Th17诱导和肠道收缩,并增加了寄生虫旋毛虫的驱逐
机译:小鼠肠道细菌调节衍生自无菌TCR-转基因小鼠的肠免疫细胞的抗原特异性细胞因子产生
机译:通过IL-13Ralpha2诱导的MAP激酶激活和活性氧的诱导,肠上皮细胞中白介素13信号的调节。
机译:R-spondin 1和Slit2诱导肠道干细胞增强了放化疗保护
机译:R-Spondin 1和SliT2增强肠杆菌诱导肠干细胞的诱导肠杆菌