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机译:绵羊PrP基因型与经典的scrapie初步传播到野生型小鼠后,与病灶和免疫组织化学模式相关。
Katy E. Beck, PhD, Rosemary E. Sallis, PhD, Richard Lockey, BSc, Marion M. Simmons, PhD, and John Spiropoulos, PhDFrom the Neuropathology Unit, Veterinary Laboratories Agency-Weybridge, New Haw, Addlestone, Surrey, United Kingdom.Send correspondence and reprint requests to: John Spiropoulos, PhD, Neuropathology Unit, Veterinary Laboratories Agency-Weybridge, New Haw, Addlestone, Surrey, KT15 3NB, United Kingdom, E-mail: j.spiropoulos@ vla.defra.gsi.gov.ukThis work was funded by the UK Department of the Environment, Food and Rural Affairs (Deffa), under projects SE1919 and SE1849.Supplemental digital content is available for this article. Direct URL citations appear in the printed text and are provided in the HTML and PDF versions of this article on the journal's Web site (www.jneuropath.com).,;
机译:羊PrP基因型与经典的瘙痒病主要传播给野生型小鼠后,与病灶和免疫组织化学模式有关。
机译:经典的瘙痒病向野生型小鼠的传播:绵羊PrP序列对病变轮廓的影响。
机译:经典瘙痒病向野生型小鼠的传播:绵羊PrP序列对病变轮廓的影响
机译:常规和绵羊PrP转基因小鼠的初级传代绵羊瘙痒病分离株的分子和传播特征
机译:常规和绵羊PrP转基因小鼠的初次传代绵羊瘙痒病分离株的分子和传播特性