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机译:PKB / Akt通过信号抑制剂LY294002介导人恶性神经胶质瘤的放射增敏作用
Departments of Radiation Oncology The University of California;
Departments of Radiation Oncology The University of California;
Departments of Radiation Oncology The University of California;
Departments of Radiation Oncology The University of California;
Brain Tumor Research Center and Neurosurgery The University of California;
Brain Tumor Research Center and Neurosurgery The University of CaliforniaCancer Research Center The University of California;
Departments of Radiation Oncology The University of CaliforniaBrain Tumor Research Center and Neurosurgery The University of CaliforniaDepartment of Radiation Oncology and Comprehensive Cancer Center UCSF;
glioma; LY294002; phosphatidylinositol 3-kinase; radiation; signal transduction;
机译:PKB / Akt在人恶性神经胶质瘤中通过信号抑制剂LY294002介导放射增敏作用。
机译:Akt信号通路:放射增敏人类恶性神经胶质瘤的目标
机译:用腺病毒介导的PTEN和PI3K抑制剂的组合治疗通过调节PI3K / AKT信号通路,体外和体内抑制恶性胶质瘤细胞生长
机译:Ras引发磷脂酰 - 肌醇-3-激酶(PI3K)/ PKB介导的信号传导途径,未转化的人外周不调T淋巴细胞
机译:多个独立于Akt / PKB的生存信号通路抑制LNCaP前列腺癌细胞的凋亡。
机译:Akt信号通路:放射增敏人类恶性神经胶质瘤的目标
机译:Akt信号通路:放射增敏人类恶性神经胶质瘤的目标