机译:插入序列ISEcp1促进质粒编码的ampCβ-内酰胺酶bla CMY-4 sub>的高水平表达,对革兰氏阴性菌具有抗性
School of Medicine and Environmental Infectious Diseases Graduate School of Medical Sciences Kitasato University 1-15-1 Kitasato Sagamihara Kanagawa 228-8555 Japan;
School of Medicine and Environmental Infectious Diseases Graduate School of Medical Sciences Kitasato University 1-15-1 Kitasato Sagamihara Kanagawa 228-8555 Japan;
School of Medicine and Environmental Infectious Diseases Graduate School of Medical Sciences Kitasato University 1-15-1 Kitasato Sagamihara Kanagawa 228-8555 Japan;
School of Medicine and Environmental Infectious Diseases Graduate School of Medical Sciences Kitasato University 1-15-1 Kitasato Sagamihara Kanagawa 228-8555 Japan;
ISEcp1; Plasmid-encoded AmpC β-lactamase; blan CMY-4; Citrobacter freundii;
机译:由于插入序列ISEcp1促进了质粒编码的ampCβ-内酰胺酶blaCMY-4的高水平表达,因此对革兰氏阴性菌具有抗性。
机译:在重症监护室患者中,延长光谱β-内酰胺酶(MBL),延长光谱β-内酰胺酶(ESBL)阳性非发酵革兰氏阴性杆菌,特别是BLA(CTX-M)和BLA的分子检测 (AMPC)基因
机译:影响表达肠杆菌来源的质粒编码ampC基因的革兰氏阴性生物的基因表达和抗性的因素。
机译:由于在大肠杆菌中-10和-35启动子序列之间插入了核苷酸而导致AmpCβ-内酰胺酶的高表达临床分离株:对广谱头孢菌素治疗无反应的病例
机译:影响基因表达和抗肠道阴性生物体的基因表达和抗性的因素,表达肠杆菌原产地的质粒编码AMPC基因