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机译:多种信号通路活性的整合解决了结肠上皮细胞对炎性细胞因子刺激的反应中的K-RAS / N-RAS突变悖论
1. Department of Biological Engineering,Massachusetts Institute of Technology, 77 Massachusetts Ave, 16-343, Cambridge, USA;
1. Molecular Pathology Unit and Center for Cancer Research,Massachusetts General Hospital, Charlestown, USA@@2. Department of Pathology,Harvard Medical School, Boston, USA;
1. Molecular Pathology Unit and Center for Cancer Research,Massachusetts General Hospital, Charlestown, USA@@2. Department of Pathology,Harvard Medical School, Boston, USA;
1. Department of Biological Engineering,Massachusetts Institute of Technology, 77 Massachusetts Ave, 16-343, Cambridge, USA@@2. Center for Cancer Research,Massachusetts Institute of Technology, Cambridge, USA;
机译:多种信号通路活性的整合解决了结肠上皮细胞对炎性细胞因子刺激的反应中的K-RAS / N-RAS突变悖论
机译:桑色素通过下调脂多糖刺激的牛乳腺上皮细胞中的核因子-κB和有丝分裂原激活的蛋白激酶(MAPK)信号通路来抑制炎症细胞因子的表达
机译:瓜尔胶纤维增加了细胞因子信号传导的抑制剂 通过Toll鈥恖ike受体2和dectin鈥表达 途径,调节小肠上皮细胞中的炎症反应
机译:CIS-9,通过NF-κB信号通路在大肠杆菌刺激后牛乳液上皮细胞对牛乳液上皮细胞的抗炎作用施加抗炎作用
机译:通过神经胶质细胞中的信号转导途径对促炎性细胞因子和药物外排转运蛋白的体外和体内调节:对HIV-1神经发病机制及其治疗的影响。
机译:在结肠上皮细胞响应炎性细胞因子刺激多种信号途径活性解析K-Ras / N-ras基因突变悖论的集成
机译:多种信号通路活动的整合解决了结肠上皮细胞对炎性细胞因子刺激反应的K-Ras / N-Ras突变悖论