机译：金属硫蛋白使GSK-3β失活可预防糖尿病相关的心脏能量代谢，炎症，亚硝基化损伤和重塑的改变
Yuehui Wang,1'2 Wenke Feng,1'2 Waiüi Xne,2 Yi Tan,1·2'3 David W. Hein,4 Xiao-Knn Li,1'5 and Lu Cai1'2'3'4From the 'Chinese-American Research Institute for Diabetic Complications, Wenzhou Medical College, Zhejiang, China, the ^Department of Medicine, University of Louisville, Louisville, Kentucky, the department of Pediatrics, University of Louisville, Louisville, Kentucky, the 4 Department of Pharmacology and Toxicology, University of Louisville, Louisville, Kentucky, and the "Engineering Research Center of Bk ireactor and Pharmaceutical Development, Jilin Agricultural University, Jilin, China.Corresponding authors: Lu Cai, 10cai001@loui_vi_le.edu, and Xiao-Kun Li, xiaokunli@163.net.Received 7 December 2008 and accepted 10 March 2009.Published ahead of print at http://diabetes.diabetesjournals.org on 26 March 2009. DOI: 10.2337/db08-1697.© 2009 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by -nc-nd/3.0/ for details.The costs of publication of this article were, defniyed in part by the payment of page charges. Tliis article must therefore Iw hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.,;
机译：金属硫蛋白使GSK-3β失活可预防糖尿病相关的心脏能量代谢，炎症，亚硝基化损伤和重塑的变化
机译：金属硫蛋白可通过调节心脏ATP合酶的硝化和失活来预防糖尿病的心脏病理变化
机译：GSK-3 Alpha Beta失活的激酶表达减轻了肥大引起的心脏纤维化，但可能允许自适应生理重塑
机译：单宁酸和过氧化氢酶中空聚合物胶囊的开发，以防止氧化应激并调节白细胞介素1β诱导的髓核炎症模型中的细胞外基质活性
机译：对纳米颗粒进行工程化处理，使其靶向递送生长因子，以防止梗塞后心脏重塑。
机译：金属硫蛋白使GSK-3β失活可预防糖尿病相关的心脏能量代谢炎症亚硝基化损伤和重塑的变化
机译：金属硫蛋白使GSK-3β失活可预防糖尿病相关的心脏能量代谢，炎症，亚硝基化损伤和重塑的变化
