机译:小鼠Smad3缺乏症可预防高脂饮食诱导的胰岛素抵抗和肥胖
Chek Kun Tan,1 Nicolas Leuenberger,2 Ming Jie Tan,1 Yew Wai Yan,1 Yinghui Chen,1 Ravi Kambadur,1 Walter Wahli,2 and Nguan Soon Tan1From the 1 School of Biological Sciences, Nanyang Technological University, Singapore, and 2 Center for Integrative Genomics, National Research Center Frontiers in Genetics, University of Lausanne, Lausanne, Switzerland.Corresponding author: Nguan Soon Tan, nstan@ntu.edu.sg.Received 10 June 2010 and accepted 25 October 2010.DOI: 10.2337/db10-0801This article contains Supplementary Data online at http://diabetes. diabelesjounials.org/lookup/suppl/doi: 1 0.2337/dbl0-080 1/-/DC1.© 2011 by lhe American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by -nc-nd/3.0/ for details.,;
机译:小鼠Smad3缺乏症可预防高脂饮食诱导的胰岛素抵抗和肥胖
机译:RGC32缺乏症可预防高脂饮食引起的肥胖和小鼠胰岛素抵抗
机译:具有超长链酰基辅酶A脱氢酶缺乏症的小鼠对高脂饮食引起的肥胖的抵抗力和胰岛素抵抗。
机译:聚合物支架可保护小鼠免受饮食引起的肥胖症和葡萄糖耐受不良。
机译:解偶联蛋白3缺乏对高脂饮食诱导肥胖和胰岛素抵抗的影响
机译:小鼠Smad3缺乏症可预防高脂饮食诱导的胰岛素抵抗和肥胖
机译:小鼠Smad3缺乏症可预防高脂饮食诱导的胰岛素抵抗和肥胖