机译:脂肪组织中c-Jun NH ^ sub 2 ^-末端激酶的选择性失活可防止饮食诱导的肥胖,并改善小鼠肝和骨骼肌的胰岛素敏感性
Xinmei Zhang,1'2 Aimin Xu,1'2'3 Sookja K. Chung,2'4 Justin H.B. Cresser,5 Gary Sweeney,5 Rachel L.C. Wong,1 Aiming Lin,6 and Karen S.L. Lam1'2From the 'Department of Medicine, Li Ka Slung Faculty of Medicine. University of Hong Kong, Hong Kong, the "Research ('enter of Heart, Brain, Hormone, and Healthy Ageing, Li Ka Siting Faculty of Medicine, University of Hong Kong, I long Kong, the ^Department of Pharmacology and Pharmacy, Li Ka Slung Faculty of Medicine, University of Hong Kong, Hong Kong, die 'Department of Anatomy, Li Ka Shing Faculty of Medicine. University of Hong Kong, Hong Kong, the "Department of Biology, York University, Toronto, Canada, and the ''Ben May Department for Cancer Research, University of Chicago, Chicago. Illinois.Corresponding author: Karen Lam. ksllam@hku.hk. or Aimin Xn. amxu@hku. hk.Received 6 May 2010 and accepted 14 November 2010.DOl: 10.2337/di) 1 0-()fi50This article contains Supplementary Data online at htlp://diabetes. diabelesjounials.org/lookup/suppl/doi:10.2337/dbl0-0650/-/DCT.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/liy -nc-nd/3.0/ for details.,;
机译:脂肪组织中c-Jun NH_2-末端激酶的选择性失活可防止饮食诱发的肥胖,并改善小鼠肝脏和骨骼肌的胰岛素敏感性
机译:肥胖小鼠中蛋白激酶Map4k4的诱导性删除可改善肝脏和脂肪组织中的胰岛素敏感性。
机译:补充谷氨酰胺可诱发饮食性肥胖大鼠的脂肪组织中的胰岛素抵抗并改善肝脏和肌肉中的胰岛素信号传导
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机译:脂肪组织中c-Jun NH2末端激酶的选择性失活可保护饮食免于肥胖并改善小鼠肝脏和骨骼肌的胰岛素敏感性
机译:脂肪组织中c-Jun NH2末端激酶的选择性失活可保护饮食免于肥胖,并改善小鼠肝脏和骨骼肌的胰岛素敏感性