机译:淀粉样蛋白β通过激活JAK2 / STAT3 / SOCS-1信号通路诱导肝胰岛素抵抗
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, China;
机译:EGR2通过用棕榈酸酯处理的HepG2细胞中通过JAK2 / Stat3 / SoCs-1途径增强胰岛素抗性
机译:高迁移率族框1诱导大鼠胰腺腺泡细胞中Janus激酶2的激活以及信号转导和转录激活子3(JAK2 / STAT3)信号通路的激活,而AG490和雷帕霉素则抑制它们的激活
机译:二十二碳烯酸对非酒精性肝脏脂肪的保护作用通过激活JAK2 / Stat3信号通路
机译:肺上皮细胞中stat3通路的过度激活引起肺部炎症和肺癌
机译:20-HETE在饮食诱导的肥胖和胰岛素抵抗中的作用:对胰岛素信号的干扰
机译:淀粉样蛋白β通过激活JAK2 / STAT3 / SOCS-1信号通路诱导肝胰岛素抵抗
机译:淀粉样蛋白通过激活JAK2 / STAT3 / SOCS-1信号通路诱导肝胰岛素抵抗力