机译:删除Rab-GTPase激活蛋白TBC14KO和TBC1D4在小鼠中消除了胰岛素和AICAR刺激的葡萄糖转运。
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany,German Center for Diabetes Research (DZD), Duesseldorf, Germany;
German Institute for Human Nutrition, Potsdam, Germany;
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany;
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany;
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany;
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany;
Department of Biology and Biochemistry, University of Bath, Bath, U.K.;
Institute of Anatomy, University of Zurich, Zurich, Switzerland;
Institute of Anatomy, University of Zurich, Zurich, Switzerland;
German Center for Diabetes Research (DZD), Duesseldorf, Germany,German Institute for Human Nutrition, Potsdam, Germany;
German Diabetes Center, Leibniz Center for Diabetes Research, Heinrich-Heine-University, Duesseldorf, Germany,German Center for Diabetes Research (DZD), Duesseldorf, Germany;
机译:常规敲除小鼠中的Tbc1d1会损害胰岛素和AICAR刺激的骨骼肌葡萄糖摄取
机译:G蛋白偶联受体30的缺失削弱了葡萄糖耐受性,降低了骨骼生长,增加了血压,并消除了雌性小鼠中雌二醇刺激的胰岛素释放。
机译:蛋白质AS160 / TBC1D4在葡萄糖向骨骼肌转运中的作用
机译:聚合物胶束上葡萄糖分子与靶向膜蛋白的葡萄糖分子的相互作用分析
机译:Nck衔接蛋白在小鼠未折叠蛋白应答和葡萄糖稳态中的功能。
机译:TBC1D1和TBC1D4在胰岛素和运动刺激的骨骼肌葡萄糖转运中的作用
机译:小鼠中Rab-GTPase激活蛋白TBC14KO和TBC1D4的删除消除了胰岛素和AICAR刺激的葡萄糖转运
机译:通过消除I131标记的蛋白质测量抗体反应。 I.从正常小鼠的血液中消除I131标记的蛋白质