首页> 外文期刊>Advances in Interventional Cardiology: Postepy w Kardiologii Interwencyjnej >Proximal end of 15-year-old ventricular electrode penetrating pulmonary tissue – a source of infection and a challenge for transvenous lead extraction
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Proximal end of 15-year-old ventricular electrode penetrating pulmonary tissue – a source of infection and a challenge for transvenous lead extraction

机译:15岁的心室电极渗透肺组织的近端 - 感染源和致乐铅提取的挑战

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We report a case of a 56-year-old man with a DDD pacemaker implanted in 1998 followed by additional ventricular lead implantation in 2000 due to lead dysfunction and battery replacement in 2006 (proximal end of the abandoned lead already observed in the pulmonary artery), with currently present clinical signs of lead-dependent infective endocarditis (LDIE). In March 2013 he was diagnosed with recurrent episodes of shortness of breath, cough and fever. Initially the patient was hospitalized in the pneumology ward and underwent a series of diagnostic examinations, including computed tomography (CT) and bronchoscopy. Chest CT revealed localized signs of inflammation (which could be differentiated from neoplastic lesions) in the mid-right pulmonary lobe. Given the vicinity of the migrated lead to the lung inflammatory process, penetration of the lead in the lung tissue was confirmed. Blood cultures were positive for Staphylococcus epidermidis (MSS). After cardiologist consultation the patient was transferred to the Cardiology Clinic with LDIE diagnosed. The patient was qualified for a transvenous lead extraction (TLE) procedure. Pre-operative transthoracic echocardiography (TTE) revealed dilatation of the right atrium and ventricle with very high pulmonary artery systolic pressure (PASP = 80 mm Hg) calculated from tricuspid regurgitation. There were no signs of vegetations in transesophageal echocardiography (TEE) examination. The TLE procedure was performed in the cardiovascular operating room with on-site surgical standby. General anesthesia was used; invasive blood pressure, ECG, and ventilation parameters were monitored. At the beginning both functional leads were removed (using a Byrd dilator mechanical sheath). After unsuccessful grasping of the non-functional lead at the level of the right ventricle (due to adherences to the wall), it was successfully captured in the pulmonary artery using a pig-tail catheter; afterwards the proximal end was grasped in the right atrium lumen with a lasso catheter and the lead was finally extracted using the left subclavian approach and dilator sheaths (Figure 1). There were no procedural or post-procedural complications. Full radiological and clinical success was achieved. Postoperative TTE/TEE showed no signs of lead fragments or vegetations and reduction of PASP (60 mm Hg). As the patient was not pace-dependent, system reimplantation was delayed. According to the current guidelines, indications for TLE of a... View full text...
机译:我们举报了一名56岁男子,一个56岁的男子,1998年植入了DDD起搏器,其次是2000年额外的心室铅植入,由于2006年的铅功能障碍和电池更换(在肺动脉中已经观察到的废弃铅的近端) ,目前存在铅依赖性感染性心内膜炎(LDIE)的临床症状。 2013年3月,他被诊断出患有呼吸急促,咳嗽和发烧的经常性剧集。最初,患者在气喘病房中住院,并经历了一系列诊断检查,包括计算断层扫描(CT)和支气管镜检查。胸部CT揭示了中右肺叶中的炎症的局部症状(可从肿瘤病变区分)。鉴于迁移的导致肺炎症过程附近,确认了肺组织中的铅的渗透。血液培养物为葡萄球菌(MSS)是阳性的。心脏病学家咨询后,患者被诊断为LDIE转移到心脏病学诊所。患者有资格用于致乐化铅提取(TLE)程序。术前经脉冲超声心动图(TTE)揭示了右心房和脑室的扩张,具有由三尖瓣重新脉冲计算的非常高的肺动脉收缩压(PASP = 80mM Hg)。在过剂声的超声心动图(TEE)检查中没有植被迹象。 TLE程序在心血管手术室中进行,现场手术备用。使用全身麻醉;侵入性血压,心电图和通风参数被监测。在开始,两种功能引线被移除(使用BYRD扩张器机械护套)。在右心室的水平下不成功掌握非功能性铅(由于粘附到墙壁),使用猪尾导管在肺动脉中成功捕获;之后,近端在右心中的内核内部粘连,套索导管,最后用左锁骨头方法和扩张器护套提取引线(图1)。没有程序性或后病症并发症。实现了全面放射和临床成功。术后TTE / TEE显示没有铅碎片或植被的迹象和覆盖物的减少(60 mm Hg)。由于患者没有依赖于步伐,系统再持续延迟。根据目前的指导方针,A的迹象是...查看全文......

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